Evidence map›Paper›PMID 33928564›Full record

ArticleMethods in molecular biology (Clifton, N.J.)2021

Murine T-Cell Transfer Colitis as a Model for Inflammatory Bowel Disease.

Patrick Maschmeyer, Jakob Zimmermann, Anja Andrea Kühl

Abstract read
PubMed Publisher
In one paragraph

Article in Methods in molecular biology (Clifton, N.J.), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. RNA Helicase DDX21 Controls CD4Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026
    Article
  2. Review
  3. Article
  4. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Patrick MaschmeyerDeutsches Rheuma-Forschungszentrum (DRFZ) Berlin, an Institute of the Leibniz Association, Berlin, Germany. Patrick.Maschmeyer@drfz.de.
Jakob ZimmermannMaurice Müller Laboratories (Department of Biomedical Research), Universitätsklinik für Viszerale Chirurgie und Medizin Inselspital, University of Bern, Bern, Switzerland.
Anja Andrea KühliPATH.Berlin - Core Unit of the Charité, Charité - Universitätsmedizin Berlin, Corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin, Germany.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Inflammatory bowel disease (IBD) is a group of severe chronic inflammatory conditions of the human gastrointestinal tract. Murine models of colitis have been invaluable tools to improve the understanding of IBD development and pathogenesis. While the disease etiology of IBD is complex and multifactorial, CD4

Indexed as

Adoptive TransferCell SeparationFlow CytometryAnimalsColitisColonDisease Models, AnimalHomeodomain ProteinsMiceMice, Inbred C57BLMice, KnockoutResearch DesignT-Lymphocytes, Helper-InducerWorkflowHomeodomain ProteinsRAG-1 proteinAdoptive transferChronic inflammationColitis modelInflammatory bowel diseaseIntestinal inflammationMouse modelT cellsT helper cellsT helper lymphocytesT lymphocytes

Identifiers

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.