Evidence map›Paper›PMID 33916643›Full record

ArticleCells2021

LPA

Silvia Anahi Valdés-Rives, Denisse Arcos-Montoya, Marisol de la Fuente-Granada, Carmen J Zamora-Sánchez, Luis Enrique Arias-Romero, Olga Villamar-Cruz, Ignacio Camacho-Arroyo, Sonia M Pérez-Tapia, Aliesha González-Arenas

Open access · goldAbstract read
In one paragraph

Article in Cells, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
1.9field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 14 citations in OpenAlex.

  1. Review
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  4. Article
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  9. Effects of progesterone on T-type-CaNeural regeneration research · 2022
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 3 institutions in 1 country.

Silvia Anahi Valdés-RivesDepartamento de Medicina Genómica y Toxicología Ambiental, Instituto de Investigaciones Biomédicas, Universidad Nacional Autónoma de México (UNAM), 04510 Ciudad de México, Mexico.ORCID 0000-0002-2104-4884
Denisse Arcos-MontoyaDepartamento de Medicina Genómica y Toxicología Ambiental, Instituto de Investigaciones Biomédicas, Universidad Nacional Autónoma de México (UNAM), 04510 Ciudad de México, Mexico.ORCID 0000-0002-6509-7662
Marisol de la Fuente-GranadaDepartamento de Medicina Genómica y Toxicología Ambiental, Instituto de Investigaciones Biomédicas, Universidad Nacional Autónoma de México (UNAM), 04510 Ciudad de México, Mexico.
Carmen J Zamora-SánchezUnidad de Investigación en Reproducción Humana, Instituto Nacional de Perinatología-Facultad de Química, Universidad Nacional Autónoma de México (UNAM), 04510 Ciudad de México, Mexico.
Luis Enrique Arias-RomeroUnidad de Investigación en Biomedicina (UBIMED), Facultad de Estudios Superiores-Iztacala, Universidad Nacional Autónoma de México (UNAM), Tlalnepantla, 54090 Estado de México, Mexico.
Olga Villamar-CruzUnidad de Investigación en Biomedicina (UBIMED), Facultad de Estudios Superiores-Iztacala, Universidad Nacional Autónoma de México (UNAM), Tlalnepantla, 54090 Estado de México, Mexico.
Ignacio Camacho-ArroyoUnidad de Investigación en Reproducción Humana, Instituto Nacional de Perinatología-Facultad de Química, Universidad Nacional Autónoma de México (UNAM), 04510 Ciudad de México, Mexico.
Sonia M Pérez-TapiaUnidad de Desarrollo e Investigación en Bioprocesos (UDIBI), Escuela Nacional de Ciencias Biológicas, Instituto Politécnico Nacional, 11350 Ciudad de México, Mexico.ORCID 0000-0002-2818-8522
Aliesha González-ArenasDepartamento de Medicina Genómica y Toxicología Ambiental, Instituto de Investigaciones Biomédicas, Universidad Nacional Autónoma de México (UNAM), 04510 Ciudad de México, Mexico.
Universidad Nacional Autónoma de México · MXInstituto Nacional de Perinatología · MXInstituto Politécnico Nacional · MX

Funding

Universidad Nacional Autónoma de México PAPIIT IA200718, IA201120
6 · The paper itself

Abstract

Lysophosphatidic acid (LPA) induces a wide range of cellular processes and its signaling is increased in several cancers including glioblastoma (GBM), a high-grade astrocytoma, which is the most common malignant brain tumor. LPA

Indexed as

Brain NeoplasmsCell Line, TumorCell MovementCell NucleusErbB ReceptorsGene Expression Regulation, NeoplasticGlioblastomaHumansLysophospholipase DLysophospholipidsPhosphoric Diester HydrolasesPhosphorylationPhosphoserinePromoter Regions, GeneticProtein BindingProtein Kinase C-alphaErbB Receptorslysophosphatidic acidLysophospholipase DLysophospholipidsPhosphoric Diester HydrolasesPhosphoserineProtein Kinase C-alphaReceptors, Lysophosphatidic AcidReceptors, ProgesteroneRNA, MessengerVascular Endothelial Growth Factor AglioblastomaLPA1 receptorprogesterone receptorprotein kinase C α

Identifiers

PMID33916643
PMCPMC8066126
OpenAlexW3151420100

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.