Evidence map›Paper›PMID 33865940›Full record

ReviewNeuroscience letters2021

Ca

Jingsong Zhou

Abstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in Neuroscience letters, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
  3. The Cellular and Molecular Signature of ALS in Muscle.Journal of personalized medicine · 2022
    Review
  4. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Jingsong ZhouDepartment of Kinesiology, College of Nursing and Health Innovation, University of Texas at Arlington, Arlington, TX 76010, USA. Electronic address: jingsong.zhou@uta.edu.

Funding

Preserving Mitochondrial Function for Alleviating ALS ProgressionR01NS105621 · NINDS · UNIVERSITY OF TEXAS ARLINGTON · PI BROTTO, MARCO, ZHOU, JINGSONG · 2019 to 2023
$2.9M
Ca signaling cross-talk from SR to mitochondria in heart muscleR01HL138570 · NHLBI · OHIO STATE UNIVERSITY · PI LIN, PEI-HUI, ZHOU, JINGSONG · 2018 to 2021
$2.6M
Ca Signaling in Progression of Amyotrophic Lateral Sclerosis in Skeletal MuscleR01AR057404 · NIAMS · RUSH UNIVERSITY MEDICAL CENTER · PI ZHOU, JINGSONG · 2010 to 2014
$1.6M
NHLBI NIH HHS R01 HL138570NIAMS NIH HHS R01 AR057404NINDS NIH HHS R01 NS105621
6 · The paper itself

Abstract

The volitional movement of skeletal is controlled by the motor neuron at the site of neuromuscular junction (NMJ) where the retrograde signals are also passed back from muscle to the motor neuron. As the normal function of muscle largely depends on mitochondria that determine the fate of a skeletal muscle myofiber, there must exist a fine-controlled functional coupling between NMJ and mitochondria in myofibers. This mini-review discusses recent publications that reveal how spatiotemporal profiles of intracellular free Ca

Indexed as

AnimalsCalciumCations, DivalentHumansMitochondriaMotor NeuronsMuscle ContractionMyofibrilsNeuromuscular JunctionCalciumCations, DivalentCa(2+) signalingMitochondriaNeuromuscular junctionSkeletal muscle

Identifiers

PMID33865940
PMCPMC8089061

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.