Evidence map›Paper›PMID 33849074›Full record

ArticleSleep2021

The natural history of insomnia: predisposing, precipitating, coping, and perpetuating factors over the early developmental course of insomnia.

Jason G Ellis, Michael L Perlis, Colin A Espie, Michael A Grandner, Célyne H Bastien, Nicola L Barclay, Ellemarije Altena, Maria Gardani

Open access · hybridAbstract read
In one paragraph

Article in Sleep, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 55 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
55citing papers in PubMed, 2 pooled it
11.9field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

55 citing papers in PubMed, 2 syntheses or guidelines pooled it, 95 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Trial
  4. Trial
  5. Article
  6. Article
  7. Article
  8. Review
  9. Article
  10. Article
  11. Article
  12. Insomnia disorder.Nature reviews. Disease primers · 2026
    Review
  13. Article
  14. Article
  15. Article
  16. Article
  17. Review
  18. Article
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 7 institutions in 4 countries.

Jason G EllisNorthumbria Sleep Research, Department of Psychology, Northumbria University, Newcastle upon Tyne, UK.ORCID 0000-0002-8496-520X
Michael L PerlisDepartment of Psychiatry, University of Pennsylvania, Philadelphia, PA.
Colin A EspieSleep and Circadian Neuroscience Institute (SCNi), University of Oxford, Oxford, UK.
Michael A GrandnerDepartment of Psychiatry, Psychology and Medicine, University of Arizona, Tucson, AZ.
Célyne H BastienSchool of Psychology, Université Laval, Quebec, Canada.
Nicola L BarclaySleep and Circadian Neuroscience Institute (SCNi), University of Oxford, Oxford, UK.
Ellemarije AltenaSchool of Psychology, University of Glasgow, Glasgow, UK.
Maria GardaniUMR 5287, Institut de Neurosciences Intégratives et Cognitives d'Aquitaine, Neuroimagerie et Cognition Humaine, CNRS, Université de Bordeaux, Bordeaux, France.
University of Oxford · GBCentre National de la Recherche Scientifique · FRNorthumbria University · GBUniversité Laval · CAUniversity of Arizona · USUniversity of Glasgow · GBUniversity of Pennsylvania · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

While there is an extensive literature on predisposing, precipitating, coping, and perpetuating factors in those with chronic insomnia, very little work has been undertaken to evaluate these factors over the early developmental course of insomnia. The present aim was to determine whether several hypothesized factors in each domain (predisposing, precipitating, coping, and perpetuating), assessed during an episode of acute insomnia (AI), are related to its persistence or remission to normal sleep. Participants comprised n = 140 people with AI and n = 737 normal sleepers (NS) recruited from the general public. Participants completed measures assessing predisposing characteristics (personality traits, arousal predisposition, and insomnia vulnerability), precipitating events and outcomes (life events, perceived stress, anxiety, and depression), coping styles (thought control strategies and coping styles), and perpetuating factors (sleep preoccupation, pre-sleep arousal, dysfunctional beliefs, and fatigue). Additionally, insomnia status (from AI at baseline to its persistence or natural remission [NR]) was assessed 1 month later (n = 129). Baseline differences between NS and individuals with AI were observed in each domain with increasing age, lower openness to experience and conscientiousness, higher insomnia severity, levels of anxiety, and affective sleep preoccupation significantly predicting AI status. Further, a previous episode of insomnia, higher depression scores, and affective sleep preoccupation scores significantly predicted its persistence, as opposed to its NR. Results are discussed with reference to the conceptualization of insomnia and how the findings may influence the design of preventative interventions to circumvent the transition from acute to chronic insomnia.

Indexed as

Sleep Initiation and Maintenance DisordersAdaptation, PsychologicalAnxietyArousalHumansSleepacute insomniadepressionpreventionsleep preoccupationSpielman

Identifiers

PMID33849074
PMCPMC8826168
OpenAlexW3152508020

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.