Evidence map›Paper›PMID 33806201›Full record

ArticleMembranes2021

Oxytocin Downregulates the Ca

Masaki Morishima, Shintaro Tahara, Yan Wang, Katsushige Ono

Open access · goldAbstract read
In one paragraph

Article in Membranes, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
2.0field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 11 citations in OpenAlex.

  1. Article
  2. Article
  3. Enhancement of CaNeuroscience bulletin · 2025
    Article
  4. Eicosapentaenoic Acid Rescues Cav1.2-L-Type CaInternational journal of molecular sciences · 2024
    Article
  5. Article
  6. Review
  7. Article
  8. Review
  9. Article
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Masaki MorishimaDepartment of Pathophysiology, Oita University School of Medicine, Oita 879-5593, Japan.
Shintaro TaharaDepartment of Pathophysiology, Oita University School of Medicine, Oita 879-5593, Japan.
Yan WangDepartment of Pathophysiology, Oita University School of Medicine, Oita 879-5593, Japan.
Katsushige OnoDepartment of Pathophysiology, Oita University School of Medicine, Oita 879-5593, Japan.
Oita University · JPKindai University · JP

Funding

Japan Society for the Promotion of Science KAKEN 21590934
6 · The paper itself

Abstract

Oxytocin (OT) and its receptor (OTR) are expressed in the heart and are involved in the physiological cardiovascular functional system. Although it is known that OT/OTR signaling is cardioprotective by reducing the inflammatory response and improving cardiovascular function, the role of OT in the cardiac electrical excitation modulation has not been clarified. This study investigates the molecular mechanism of the action of OT on cardiomyocyte membrane excitation focusing on the L-type Ca

Indexed as

CREBgene transcriptionL-type Ca2+ channeloxytocin

Identifiers

PMID33806201
PMCPMC8066716
OpenAlexW3138773793

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.