Evidence map›Paper›PMID 33804408›Full record

ReviewProteomes2021

C1q Complement/Tumor Necrosis Factor-Associated Proteins in Cardiovascular Disease and COVID-19.

Yaoli Xie, Zhijun Meng, Jia Gao, Caihong Liu, Jing Wang, Rui Guo, Jianli Zhao, Bernard Lopez, Theodore Christopher, Daniel Lee and 2 more

Open access · goldAbstract readReview
In one paragraph

Review in Proteomes, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
1.4field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 12 citations in OpenAlex.

  1. Unveiling the roles of CTRP family in cardiac remodeling.Journal of molecular medicine (Berlin, Germany) · 2025
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 2 institutions in 2 countries.

Yaoli XieDepartment of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA 19107, USA.ORCID 0000-0002-0110-8756
Zhijun MengDepartment of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA 19107, USA.
Jia GaoDepartment of Physiology, Shanxi Medical University, Taiyuan 030001, China.ORCID 0000-0002-5966-6312
Caihong LiuDepartment of Physiology, Shanxi Medical University, Taiyuan 030001, China.
Jing WangDepartment of Physiology, Shanxi Medical University, Taiyuan 030001, China.
Rui GuoDepartment of Physiology, Shanxi Medical University, Taiyuan 030001, China.
Jianli ZhaoDepartment of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA 19107, USA.
Bernard LopezDepartment of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA 19107, USA.
Theodore ChristopherDepartment of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA 19107, USA.
Daniel LeeDepartment of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA 19107, USA.
Xinliang MaDepartment of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA 19107, USA.ORCID 0000-0002-9041-0876
Yajing WangDepartment of Emergency Medicine, Thomas Jefferson University, Philadelphia, PA 19107, USA.ORCID 0000-0002-6358-6948
Thomas Jefferson University · USShanxi Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

With continually improving treatment strategies and patient care, the overall mortality of cardiovascular disease (CVD) has been significantly reduced. However, this success is a double-edged sword, as many patients who survive cardiovascular complications will progress towards a chronic disorder over time. A family of adiponectin paralogs designated as C1q complement/tumor necrosis factor (TNF)-associated proteins (CTRPs) has been found to play a role in the development of CVD. CTRPs, which are comprised of 15 members, CTRP1 to CTRP15, are secreted from different organs/tissues and exhibit diverse functions, have attracted increasing attention because of their roles in maintaining inner homeostasis by regulating metabolism, inflammation, and immune surveillance. In particular, studies indicate that CTRPs participate in the progression of CVD, influencing its prognosis. This review aims to improve understanding of the role of CTRPs in the cardiovascular system by analyzing current knowledge. In particular, we examine the association of CTRPs with endothelial cell dysfunction, inflammation, and diabetes, which are the basis for development of CVD. Additionally, the recently emerged novel coronavirus (COVID-19), officially known as severe acute respiratory syndrome-coronavirus-2 (SARS-CoV-2), has been found to trigger severe cardiovascular injury in some patients, and evidence indicates that the mortality of COVID-19 is much higher in patients with CVD than without CVD. Understanding the relationship of CTRPs and the SARS-CoV-2-related damage to the cardiovascular system, as well as the potential mechanisms, will achieve a profound insight into a therapeutic strategy to effectively control CVD and reduce the mortality rate.

Indexed as

cardiovascular diseaseCOVID-19CTRPsobesity

Identifiers

PMID33804408
PMCPMC7931048
OpenAlexW3135430183

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.