Evidence map›Paper›PMID 33778919›Full record

ArticleMolecular biology reports2021

Nitric oxide synthase-2 (NOS2) gene polymorphism c.1832C>T (Ser608Leu) associated with nitrosative stress in Fanconi anaemia.

Merin George, Avani Solanki, Purvi Mohanty, Somprakash Dhangar, Aruna Rajendran, Sheila Mohan, Babu Rao Vundinti

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In one paragraph

Article in Molecular biology reports, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.2field-weighted citation impact, top 48% of its field
1 · What the graph read from it

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3 · Its place in the literature

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1 citing paper in PubMed, 2 citations in OpenAlex.

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4 · The record

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5 · Who and what money

Authors and funding

7 authors at 3 institutions in 1 country.

Merin GeorgeDepartment of Cytogenetics, ICMR-National Institute of Immunohematology, K. E. M. Hospital Campus, Parel, Mumbai, 400 012, Maharashtra, India.
Avani SolankiDepartment of Cytogenetics, ICMR-National Institute of Immunohematology, K. E. M. Hospital Campus, Parel, Mumbai, 400 012, Maharashtra, India.
Purvi MohantyDepartment of Cytogenetics, ICMR-National Institute of Immunohematology, K. E. M. Hospital Campus, Parel, Mumbai, 400 012, Maharashtra, India.
Somprakash DhangarDepartment of Cytogenetics, ICMR-National Institute of Immunohematology, K. E. M. Hospital Campus, Parel, Mumbai, 400 012, Maharashtra, India.
Aruna RajendranDepartment of Paediatric Haematology, Institute of Child Health and Hospital for Children, Chennai, India.
Sheila MohanDepartment of Paediatric Haematology, Oncology, Apollo Speciality Hospital, Chennai, India.
Babu Rao VundintiDepartment of Cytogenetics, ICMR-National Institute of Immunohematology, K. E. M. Hospital Campus, Parel, Mumbai, 400 012, Maharashtra, India. vbaburao@hotmail.com.ORCID http://orcid.org/0000-0003-4783-2691
ICMR - National Institute of Immunohaemotology · INApollo Hospitals · INInstitute of Child Health · IN

Funding

Science and Engineering Research Board EEQ/2016/000510
6 · The paper itself

Abstract

Fanconi anemia (FA) occurs due to genomic instability with predisposition to bone marrow failure, phenotypic abnormalities and cancers. Though mutations in 22 genes leading to DNA repair defect have been identified, the cellular factor such as oxidative stress has also shown to be associated with FA. Nitrosative Stress (NS) is biochemically correlated to many oxidative stress related disorders and the NS as a pathological hallmark in FA has been so far overlooked. We carried out the study first time in Indian patients with FA with an objective to understand the role of NS in the pathogenesis of FA. The study was carried out in 70 FA subjects. The FA subjects were diagnosed by chromosomal breakage analysis. Molecular study was carried out by Next Generation Sequencing and Sanger sequencing. The 3-nitrotyrosine [3-NT] levels were estimated through enzyme-linked immuno-sorbent assay (ELISA) and the nitric oxide synthase genes- NOS1 (c.-420-34221G>A (rs1879417), c.-420-10205C>T (rs499776), c.4286+720G>C (rs81631)) and NOS2 (c.1823C>T (p. Ser608Leu) (rs2297518)) polymorphism were studied by direct sequencing. Chromosomal breakage analysis revealed a high frequency of chromosomal breaks (Mean chromosomal breakage-4.13 ± 1.5 breaks/metaphase) in 70 FA patients as compared to the control. Molecular studies revealed FANCA (58.34%), FANCG (18.34%) and FANCL (16.6%) complementation groups. The 3-nitrotyrosine [3-NT] levels showed to be significantly (p < 0.05) elevated in FA subjects when compared to the age match controls. Genotyping of the NOS2 gene c.1823C>T (p. Ser608Leu) (rs2297518), showed statistically significant (P < 0.05) association with FA. Elevated level of 3-NT is one of the cause of NS and NOS2 gene polymorphism associated with FA is an important target in the treatment regimen.

Indexed as

Genetic Association StudiesAdolescentAdultBase SequenceCase-Control StudiesChildChild, PreschoolFanconi AnemiaGene FrequencyHumansNitric Oxide Synthase Type INitric Oxide Synthase Type IINitrosative StressPolymorphism, Single NucleotideTyrosineYoung Adult3-nitrotyrosineNitric Oxide Synthase Type INitric Oxide Synthase Type IINOS1 protein, humanNOS2 protein, humanTyrosine3-NitrotyrosineChromosomal breakagesComplementation groupsNitrosative stressNOS1NOS2 gene polymorphismOxidative stress

Identifiers

PMID33778919
OpenAlexW3150099590

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.