Evidence map›Paper›PMID 33572816›Full record

ArticleInternational journal of molecular sciences2021

An Adult Mouse Model of Dilated Cardiomyopathy Caused by Inducible Cardiac-Specific

Hye Hyeon Yun, Soon Young Jung, Bong Woo Park, Ji Seung Ko, Kyunghyun Yoo, Jiyoung Yeo, Hong Lim Kim, Hun Jun Park, Ho Joong Youn, Jeong Hwa Lee

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
0.6field-weighted citation impact, top 31% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 6 citations in OpenAlex.

  1. Depletion of BIS in astrocytes aggravates reactive gliosis following photothrombotic brain injury.The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology · 2026
    Article
  2. Article
  3. Article
  4. Skeletal Muscle-SpecificInternational journal of molecular sciences · 2023
    Article
  5. Article
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

Hye Hyeon YunDepartment of Biochemistry, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.
Soon Young JungDepartment of Biochemistry, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.
Bong Woo ParkDepartment of Internal Medicine, Division of Cardiology, Seoul St. Mary's Hospital, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.
Ji Seung KoLaboratory Animal Research Center, Institute of Biomedical Industry, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.
Kyunghyun YooDepartment of Biochemistry, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.
Jiyoung YeoDepartment of Biochemistry, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.
Hong Lim KimIntegrative Research Support Center, Laboratory of Electron Microscope, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.
Hun Jun ParkDepartment of Internal Medicine, Division of Cardiology, Seoul St. Mary's Hospital, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.ORCID 0000-0001-8009-9546
Ho Joong YounDepartment of Internal Medicine, Division of Cardiology, Seoul St. Mary's Hospital, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.
Jeong Hwa LeeDepartment of Biochemistry, College of Medicine, The Catholic University of Korea, Seoul 16591, Korea.ORCID 0000-0002-5970-194X
Catholic University of Korea · KRThe Catholic University of Korea Seoul St. Mary's Hospital · KR

Funding

Seoul St. Mary's Hospital, The Catholic University of Korea Research Fundthe Ministry of Health and Welfare, Korea HI17C1257the National Research Foundation of Korea NRF-2019R1A2C1086949
6 · The paper itself

Abstract

BCL-2 interacting cell death suppressor (BIS) is a multifunctional protein that has been implicated in cancer and myopathy. Various mutations of the

Indexed as

Adaptor Proteins, Signal TransducingAnimalsApoptosis Regulatory ProteinsCardiomyopathy, DilatedDisease Models, AnimalFemaleGene DeletionGene Knockout TechniquesMaleMiceMice, KnockoutMyocardiumAdaptor Proteins, Signal TransducingApoptosis Regulatory ProteinsBag3 protein, mouseBISdilated cardiomyopathytamoxifen

Identifiers

PMID33572816
PMCPMC7866246
OpenAlexW3128638036

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.