Evidence map›Paper›PMID 33567693›Full record

ArticleCancers2021

Tuberculous Fibrosis Enhances Tumorigenic Potential via the NOX4-Autophagy Axis.

Seong Ji Woo, Youngmi Kim, Harry Jung, Jae Jun Lee, Ji Young Hong

Open access · goldAbstract read
In one paragraph

Article in Cancers, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
1.8field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 24 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Article
  5. Tuberculous pleural effusion-induced Arg-1Respiratory research · 2024
    Article
  6. Increased Lung Cancer Risk and Associated Risk Factors in Tuberculosis Survivors: A Korean Population-Based Study.Clinical infectious diseases : an official publication of the Infectious Diseases Society of America · 2023
    Article
  7. Review
  8. MicroRNA 148a Suppresses Tuberculous Fibrosis by Targeting NOX4 and POLDIP2.International journal of molecular sciences · 2022
    Article
  9. Review
  10. Advances in molecular mechanisms of interaction betweenTranslational lung cancer research · 2021
    Review
  11. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Seong Ji WooInstitute of New Frontier Research, Hallym University College of Medicine, Chuncheon, Gangwon-do 24253, Korea.
Youngmi KimInstitute of New Frontier Research, Hallym University College of Medicine, Chuncheon, Gangwon-do 24253, Korea.
Harry JungInstitute of New Frontier Research, Hallym University College of Medicine, Chuncheon, Gangwon-do 24253, Korea.ORCID 0000-0002-7691-8403
Jae Jun LeeInstitute of New Frontier Research, Hallym University College of Medicine, Chuncheon, Gangwon-do 24253, Korea.
Ji Young HongInstitute of New Frontier Research, Hallym University College of Medicine, Chuncheon, Gangwon-do 24253, Korea.ORCID 0000-0002-3132-7706
Hallym University · KR

Funding

National Research Foundation (NRF) funded by the Korean government NRF 2020R1A2C1011455National Research Foundation of Korea Grant funded by Korean Government 2018R1D1A1B07051017
6 · The paper itself

Abstract

While a higher incidence of lung cancer in subjects with previous tuberculous infection has been reported in epidemiologic data, the mechanism by which previous tuberculosis affects lung cancer remains unclear. We investigated the role of NOX4 in tuberculous pleurisy-assisted tumorigenicity both in vitro and in vivo.Heat-killed

Indexed as

autophagylung cancerNOX4tuberculous fibrosis

Identifiers

PMID33567693
PMCPMC7916030
OpenAlexW3127204566

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.