Evidence map›Paper›PMID 33553219›Full record

ArticleFrontiers in medicine2020

Can GPR4 Be a Potential Therapeutic Target for COVID-19?

Li V Yang, Karen A Oppelt, Mary Jane Thomassen, Mona A Marie, Shayan Nik Akhtar, Justin D McCallen

Open access · goldAbstract read
In one paragraph

Article in Frontiers in medicine, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.8field-weighted citation impact, top 24% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 16 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Li V YangDepartment of Internal Medicine, Brody School of Medicine, East Carolina University, Greenville, NC, United States.
Karen A OppeltDepartment of Comparative Medicine, Brody School of Medicine, East Carolina University, Greenville, NC, United States.
Mary Jane ThomassenDepartment of Internal Medicine, Brody School of Medicine, East Carolina University, Greenville, NC, United States.
Mona A MarieDepartment of Internal Medicine, Brody School of Medicine, East Carolina University, Greenville, NC, United States.
Shayan Nik AkhtarDepartment of Anatomy and Cell Biology, Brody School of Medicine, East Carolina University, Greenville, NC, United States.
Justin D McCallenDepartment of Internal Medicine, Brody School of Medicine, East Carolina University, Greenville, NC, United States.
East Carolina University · US

Funding

Role of the GPR4 Receptor in Intestinal Inflammation and CancerR15DK109484 · NIDDK · EAST CAROLINA UNIVERSITY · PI YANG, LI · 2016 to 2016
$436k
NIDDK NIH HHS R15 DK109484
6 · The paper itself

Abstract

Coronavirus disease 19 (COVID-19), caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), first emerged in late 2019 and has since rapidly become a global pandemic. SARS-CoV-2 infection causes damages to the lung and other organs. The clinical manifestations of COVID-19 range widely from asymptomatic infection, mild respiratory illness to severe pneumonia with respiratory failure and death. Autopsy studies demonstrate that diffuse alveolar damage, inflammatory cell infiltration, edema, proteinaceous exudates, and vascular thromboembolism in the lung as well as extrapulmonary injuries in other organs represent key pathological findings. Herein, we hypothesize that GPR4 plays an integral role in COVID-19 pathophysiology and is a potential therapeutic target for the treatment of COVID-19. GPR4 is a pro-inflammatory G protein-coupled receptor (GPCR) highly expressed in vascular endothelial cells and serves as a "gatekeeper" to regulate endothelium-blood cell interaction and leukocyte infiltration. GPR4 also regulates vascular permeability and tissue edema under inflammatory conditions. Therefore, we hypothesize that GPR4 antagonism can potentially be exploited to mitigate the hyper-inflammatory response, vessel hyper-permeability, pulmonary edema, exudate formation, vascular thromboembolism and tissue injury associated with COVID-19.

Indexed as

COVID-19endothelial cellGPR4inflammationthromboembolismvascular permeability

Identifiers

PMID33553219
PMCPMC7859652
OpenAlexW3124134321

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.