Evidence map›Paper›PMID 33516393›Full record

ReviewCardiac electrophysiology clinics2021

Atrial Fibrillation in Inherited Channelopathies.

Baha'a Al-Azaam, Dawood Darbar

Open access · greenAbstract readReview
In one paragraph

Review in Cardiac electrophysiology clinics, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed, 1 pooled it
1.1field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 1 synthesis or guideline pooled it, 9 citations in OpenAlex.

  1. Guideline
  2. Supraventricular tachycardias in ion channel diseases.Herzschrittmachertherapie & Elektrophysiologie · 2026
    Review
  3. Review
  4. Human Genetics of Cardiac Arrhythmias.Advances in experimental medicine and biology · 2024
    Review
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 1 country.

Baha'a Al-AzaamDivision of Cardiology, Department of Medicine, University of Illinois at Chicago, 820 S Wood Street, Suite 920S, Chicago, IL 60612, USA; Division of Cardiology, Department of Pharmacology, University of Illinois at Chicago, 820 S Wood Street, Suite 920S, Chicago, IL 60612, USA.
Dawood DarbarDivision of Cardiology, Department of Medicine, University of Illinois at Chicago, 820 S Wood Street, Suite 920S, Chicago, IL 60612, USA; Division of Cardiology, Department of Pharmacology, University of Illinois at Chicago, 820 S Wood Street, Suite 920S, Chicago, IL 60612, USA; Department of Medicine, Jesse Brown Veterans Administration, 820 S Wood Street, Suite 920S, Chicago, IL 60612, USA. Electronic address: Darbar@uic.edu.
Jesse Brown VA Medical Center · USUniversity of Illinois Chicago · US

Funding

Human iPSC-derived atrial cardiomyocytes to model atrial fibrillation in a dishR01HL150586 · NHLBI · UNIVERSITY OF ILLINOIS AT CHICAGO · PI DARBAR, DAWOOD, KHETANI, SALMAN R · 2020 to 2023
$2.6M
Deciphering the genetic mechanisms of atrial fibrillationR01HL138737 · NHLBI · UNIVERSITY OF ILLINOIS AT CHICAGO · PI DARBAR, DAWOOD · 2017 to 2020
$2.6M
Training Program in Personalized Cardiovascular Medicine (TPIPCVM)T32HL139439 · NHLBI · UNIVERSITY OF ILLINOIS AT CHICAGO · PI DARBAR, DAWOOD · 2018 to 2022
$2.1M
BLRD VA I01 BX004268NHLBI NIH HHS R01 HL138737NHLBI NIH HHS R01 HL150586NHLBI NIH HHS T32 HL139439
6 · The paper itself

Abstract

Atrial fibrillation (AF), the common sustained arrhythmia in clinical practice, has major public health implications due to its associated morbidity and increased mortality. The AF epidemic is due to the burgeoning elderly population and the identification of novel risk factors, for example, genetics. Since the diagnosis of AF has a major impact on the clinical assessment and management of patients with inherited arrhythmia syndromes, improved understanding of the cause and pathogenesis of AF has provided important insights into the underlying pathophysiological mechanisms of this common arrhythmia and identified potential mechanism-based therapies.

Indexed as

Atrial FibrillationChannelopathiesArrhythmias, CardiacHumansAtrial fibrillationBrugada syndromeChannelopathiesEarly repolarizationsLong QT syndromeShort QT syndrome

Identifiers

PMID33516393
PMCPMC9172630
OpenAlexW3120676593

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.