Evidence map›Paper›PMID 33487099›Full record

ArticleAutophagy2021

ATI1 (ATG8-interacting protein 1) and ATI2 define a plant starvation-induced reticulophagy pathway and serve as MSBP1/MAPR5 cargo receptors.

Jian Wu, Simon Michaeli, Lorenzo Picchianti, Yasin Dagdas, Gad Galili, Hadas Peled-Zehavi

Open access · bronzeAbstract read
In one paragraph

Article in Autophagy, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 36 papers.

0numbers the graph read from it
0cells of the map it votes in
36citing papers in PubMed
4.9field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

36 citing papers in PubMed, 56 citations in OpenAlex.

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  19. Autophagy in plants.Autophagy reports · 2024
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 2 countries.

Jian WuDepartment of Plant and Environmental Sciences, Weizmann Institute of Science, Rehovot, Israel.ORCID 0000-0001-5276-8271
Simon MichaeliDepartment of Plant and Environmental Sciences, Weizmann Institute of Science, Rehovot, Israel.ORCID 0000-0003-0123-6544
Lorenzo PicchiantiGregor Mendel Institute (GMI), Austrian Academy of Sciences, Vienna BioCenter (VBC), Vienna, Austria.
Yasin DagdasGregor Mendel Institute (GMI), Austrian Academy of Sciences, Vienna BioCenter (VBC), Vienna, Austria.ORCID 0000-0002-9502-355X
Gad GaliliDepartment of Plant and Environmental Sciences, Weizmann Institute of Science, Rehovot, Israel.
Hadas Peled-ZehaviDepartment of Plant and Environmental Sciences, Weizmann Institute of Science, Rehovot, Israel.ORCID 0000-0001-8169-5104
Weizmann Institute of Science · ILGregor Mendel Institute of Molecular Plant Biology · AT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Reticulophagy, the selective autophagy of endoplasmic reticulum (ER) components, is known to operate in eukaryotes from yeast and unicellular algae to animals and plants. Thus far, only ER-stress induced reticulophagy was reported and analyzed in plants. In this study we characterize a reticulophagy pathway in

Indexed as

ArabidopsisArabidopsis ProteinsAutophagyAutophagy-Related Protein 8 FamilyCarrier ProteinsEndoplasmic ReticulumEndoplasmic Reticulum StressMembrane ProteinsPlants, Genetically ModifiedProgesterone-Binding GlobulinProteolysisVesicular Transport ProteinsArabidopsis ProteinsATG8 protein, ArabidopsisATI1 protein, ArabidopsisAutophagy-Related Protein 8 FamilyCarrier ProteinsMembrane ProteinsMSBP1 protein, ArabidopsisProgesterone-Binding GlobulinVesicular Transport ProteinsArabidopsis thalianaautophagycargo receptorER-Phagyorganelle degradationreticulophagyTOR

Identifiers

PMID33487099
PMCPMC8632275
OpenAlexW3125071347

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.