Article in Scientific reports, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.8field-weighted citation impact, top 27% of its field
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literature
Who cites it
5 citing papers in PubMed, 7 citations in OpenAlex.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
9 authors at 3 institutions in 1 country.
Philipp Boehm-SturmDepartment of Experimental Neurology and Center for Stroke Research Berlin, Charité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin, Germany. philipp.boehm-sturm@charite.de.ORCID https://orcid.org/0000-0001-8777-4823
Susanne MuellerDepartment of Experimental Neurology and Center for Stroke Research Berlin, Charité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin, Germany.ORCID https://orcid.org/0000-0002-5053-2211
Nancy FreitagDepartment for Obstetrics and Fetal Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
Sophia BorowskiDepartment for Obstetrics and Fetal Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
Marco FoddisDepartment of Experimental Neurology and Center for Stroke Research Berlin, Charité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin, Germany.
Stefan P KochDepartment of Experimental Neurology and Center for Stroke Research Berlin, Charité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin, Germany.ORCID https://orcid.org/0000-0001-6606-6369
Sebastian TemmeDepartment of Molecular Cardiology, Heinrich-Heine-University of Düsseldorf, Düsseldorf, Germany.
Ulrich FlögelDepartment of Molecular Cardiology, Heinrich-Heine-University of Düsseldorf, Düsseldorf, Germany.
Sandra M BloisDepartment for Obstetrics and Fetal Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany. s.blois@uke.de.
Humboldt-Universität zu Berlin · DEUniversität Hamburg · DEHeinrich Heine University Düsseldorf · DE
Funding
Deutsche Forschungsgemeinschaft BL-1115/4-1Deutsche Forschungsgemeinschaft BL-1115/7-1Deutsche Forschungsgemeinschaft BO 4484/2-1Deutsche Forschungsgemeinschaft FL303/6-1Deutsche Forschungsgemeinschaft SFB1116Deutsche Forschungsgemeinschaft TE1209/1-1Deutsche Forschungsgemeinschaft TRR259Deutscher Akademischer Austauschdienst PKZ 91684410Else Kröner-Fresenius-Stiftung (2017_A123)European Research Council NOVA-MRI and PRISAR2
6 · The paper itself
Abstract
Placental hypoperfusion and hypoxia are key drivers in complications during fetal development such as fetal growth restriction and preeclampsia. In order to study the mechanisms of disease in mouse models, the development of quantitative biomarkers of placental hypoxia is a prerequisite. The goal of this exploratory study was to establish a technique to noninvasively characterize placental partial pressure of oxygen (PO
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Phenotyping placental oxygenation in Lgals1 deficient mice using · full record | OpenQuestion