Evidence map›Paper›PMID 33478328›Full record

ArticleRNA biology2021

Hypoxia activated long non-coding RNA HABON regulates the growth and proliferation of hepatocarcinoma cells by binding to and antagonizing HIF-1 alpha.

Cheng-Ning Ma, Lu-Lu Wo, Di-Fei Wang, Ci-Xiang Zhou, Jing-Chi Li, Xin Zhang, Xiu-Feng Gong, Chen-Long Wang, Ming He, Qian Zhao

Open access · greenAbstract read
In one paragraph

Article in RNA biology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
1.0field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 14 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 3 institutions in 1 country.

Cheng-Ning MaDepartment of Pathophysiology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University, School of Medicine, Research Units of Stress and Tumor, Chinese Academy of Medical Sciences, Shanghai, China.
Lu-Lu WoDepartment of Pathophysiology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University, School of Medicine, Research Units of Stress and Tumor, Chinese Academy of Medical Sciences, Shanghai, China.
Di-Fei WangDepartment of Pathophysiology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University, School of Medicine, Research Units of Stress and Tumor, Chinese Academy of Medical Sciences, Shanghai, China.
Ci-Xiang ZhouDepartment of Pathophysiology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University, School of Medicine, Research Units of Stress and Tumor, Chinese Academy of Medical Sciences, Shanghai, China.
Jing-Chi LiDepartment of Pathophysiology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University, School of Medicine, Research Units of Stress and Tumor, Chinese Academy of Medical Sciences, Shanghai, China.
Xin ZhangDepartment of Pathophysiology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University, School of Medicine, Research Units of Stress and Tumor, Chinese Academy of Medical Sciences, Shanghai, China.
Xiu-Feng GongDepartment of Pathophysiology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University, School of Medicine, Research Units of Stress and Tumor, Chinese Academy of Medical Sciences, Shanghai, China.
Chen-Long WangDepartment of Pathology, Xuzhou Medical University, Xuzhou, China.
Ming HeDepartment of Pathophysiology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University, School of Medicine, Research Units of Stress and Tumor, Chinese Academy of Medical Sciences, Shanghai, China.
Qian ZhaoDepartment of Pathophysiology, Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Shanghai Jiao Tong University, School of Medicine, Research Units of Stress and Tumor, Chinese Academy of Medical Sciences, Shanghai, China.
Chinese Academy of Medical Sciences & Peking Union Medical College · CNShanghai Jiao Tong University · CNXuzhou Medical College · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The adaptation of tumour cells to hypoxic microenvironment is one of the most significant characteristics of many malignant tumour diseases including hepatocarcinoma. Recently, long non-coding RNAs (lncRNAs) have been reported to play important roles in the various levels of gene regulation thus functioning in growth and survival of tumour cells. Here, new hypoxia-related lncRNAs in hepatocarcinoma cells were screened and validated by lncRNA chip-array as well as real-time RT-PCR. Among them, a hypoxia-activated lncRNA that we identified and termed Hypoxia-Activated BNIP3 Overlapping Non-coding RNA (HABON), was not only regulated by hypoxic-induced factor-1α (HIF-1α) but its expression increased significantly under hypoxia in tumour cells. We deciphered the biological characteristics of HABON including its cell localization, genomic location, as well as its full-length sequence, and proved HABON could promote growth, proliferation and clone-formation of hepatocarcinoma cells under hypoxia. Then, we revealed that HABON was transcriptionally activated by HIF-1α in hypoxic cells, furthermore, it could interact with HIF-1α and promote its protein degradation, thus affecting transcription of HIF-1α's target genes to exert its effects on cells. Besides, the elevated expression of HABON under hypoxia could promote the transcriptional activation of BNIP3 through HIF-1α, and increasing the expression level of BNIP3. This research provides a novel clue for the adaptive survival and growth mechanism of tumour under hypoxia, and gives a way to reveal the nature of tumour cells' resistance characteristics to harsh microenvironment.

Indexed as

Gene Expression Regulation, NeoplasticApoptosisCarcinoma, HepatocellularCell CommunicationCell ProliferationHumansHypoxiaHypoxia-Inducible Factor 1, alpha SubunitLiver NeoplasmsRNA, Long NoncodingTumor Cells, CulturedTumor MicroenvironmentHIF1A protein, humanHypoxia-Inducible Factor 1, alpha SubunitRNA, Long NoncodingBNIP3HABONHepatocarcinomaHIF-1αhypoxialncRNA

Identifiers

PMID33478328
PMCPMC8583305
OpenAlexW3125820907

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.