Evidence map›Paper›PMID 33469833›Full record

ArticleClinical reviews in allergy & immunology2021

In Search of an Association Between Genotype and Phenotype in Hereditary Angioedema due to C1-INH Deficiency.

David Loli-Ausejo, Alberto López-Lera, Christian Drouet, Marina Lluncor, Elsa Phillips-Anglés, María Pedrosa, Rosario Cabañas, Teresa Caballero

Abstract readMulticenter Study
PubMed Publisher
In one paragraph

Article in Clinical reviews in allergy & immunology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
0.9field-weighted citation impact, top 26% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 9 citations in OpenAlex.

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  9. Frontiers in allergy · 2022
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 2 countries.

David Loli-AusejoAllergy Department, Hospital Universitario La Paz, Madrid, Spain. David.loli.ausejo@gmail.com.ORCID http://orcid.org/0000-0002-6282-0122
Alberto López-LeraHospital La Paz Institute for Health Research (IdiPaz), Madrid, Spain.ORCID http://orcid.org/0000-0002-9596-6910
Christian DrouetInserm U1016, CNRS UMR8104, Institut Cochin, Université de Paris, Paris, France.ORCID http://orcid.org/0000-0003-1318-4278
Marina LluncorAllergy Department, Hospital Universitario La Paz, Madrid, Spain.ORCID http://orcid.org/0000-0003-3718-650X
Elsa Phillips-AnglésAllergy Department, Hospital Universitario La Paz, Madrid, Spain.ORCID http://orcid.org/0000-0002-9939-4163
María PedrosaAllergy Department, Hospital Universitario La Paz, Madrid, Spain.ORCID http://orcid.org/0000-0002-5295-870X
Rosario CabañasAllergy Department, Hospital Universitario La Paz, Madrid, Spain.ORCID http://orcid.org/0000-0001-8601-1728
Teresa CaballeroAllergy Department, Hospital Universitario La Paz, Madrid, Spain.ORCID http://orcid.org/0000-0003-3005-9858
Hospital Universitario La Paz · ESCentre National de la Recherche Scientifique · FRHospital La Paz Institute for Health Research · ES

Funding

Instituto de Salud Carlos III (ISCIII, Ministerio de Economía y Competitividad) and Fondos FEDER PI15-00255
6 · The paper itself

Abstract

Hereditary angioedema due to C1 inhibitor deficiency (C1-INH-HAE) is caused by mutations affecting the SERPING1 gene. Adult patients (≥ 18 years old) diagnosed with C1-INH-HAE were clustered according to a modified SERPING1 gene mutation classification [5]. Demographic, clinical, and laboratory data were studied. Published manuscripts on the genotype/phenotype relationship were reviewed. Eighty-eight patients participated in the study, with 78 having a classifiable mutation. We compared the data in the 3 largest groups: class 0 only (n = 32), class II only (n = 18), class III only (n = 22). Antigenic C4 and C1 inhibitors were higher in class II (p = 0.008 and p = 0.02, respectively), and facial attacks in the last 6 months were more frequent in class III (p = 0.04)). All the other differences were non-significant. Twelve manuscripts on phenotype/genotype correlation were found: missense mutations in SERPING1 gene were associated with delay in disease onset and lower severity score in some studies, whereas the CC F12-C46T/C polymorphism produced earlier disease onset. Our study shows minimal differences regarding clinical phenotype in patients with class 0, II, and III SERPING1 gene mutations, with a tendency to class III having a more severe phenotype. The study should be performed in a larger sample to confirm if they are significant.We propose that larger multicenter, international studies are performed, comparing different SERPING1 gene mutation classifications, combining polymorphisms in other involved genes (kallikrein-kinin system, regulation of vasculature, plasminogen activation) and using different variables and clinical scores to assess C1-INH-HAE disease activity and/or severity in order to study possible genotype/phenotype relationships.

Indexed as

Angioedemas, HereditaryAdolescentComplement C1 Inhibitor ProteinGenetic Association StudiesGenotypeHumansPhenotypeComplement C1 Inhibitor ProteinAssociationGenotypeHereditary angioedemaPhenotypeSERPING1 gene

Identifiers

PMID33469833
OpenAlexW3123186927

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.