Evidence map›Paper›PMID 33466875›Full record

ArticleCells2021

Alteration of Neural Stem Cell Functions in Ataxia and Male Sterility Mice: A Possible Role of β-Tubulin Glutamylation in Neurodegeneration.

Abdullah Md Sheikh, Shozo Yano, Shatera Tabassum, Koji Omura, Asuka Araki, Shingo Mitaki, Yoshie Ito, Shuai Huang, Atsushi Nagai

Open access · goldAbstract read
In one paragraph

Article in Cells, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.5field-weighted citation impact, top 39% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 6 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Review
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 2 countries.

Abdullah Md SheikhDepartment of Laboratory Medicine, Shimane University Faculty of Medicine, 89-1 Enya Cho, Izumo 693-8501, Japan.ORCID 0000-0003-1221-7787
Shozo YanoDepartment of Laboratory Medicine, Shimane University Faculty of Medicine, 89-1 Enya Cho, Izumo 693-8501, Japan.
Shatera TabassumDepartment of Laboratory Medicine, Shimane University Faculty of Medicine, 89-1 Enya Cho, Izumo 693-8501, Japan.
Koji OmuraDepartment of Organ Pathology, Shimane University Faculty of Medicine, 89-1 Enya Cho, Izumo 693-8501, Japan.
Asuka ArakiDepartment of Organ Pathology, Shimane University Faculty of Medicine, 89-1 Enya Cho, Izumo 693-8501, Japan.ORCID 0000-0002-2185-4828
Shingo MitakiDepartment of Neurology, Shimane University Faculty of Medicine, 89-1 Enya Cho, Izumo 693-8501, Japan.
Yoshie ItoDepartment of Neurology, Shimane University Faculty of Medicine, 89-1 Enya Cho, Izumo 693-8501, Japan.
Shuai HuangDepartment of Orthopedic Surgery, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgangdong Road, Guangzhou 510260, China.
Atsushi NagaiDepartment of Neurology, Shimane University Faculty of Medicine, 89-1 Enya Cho, Izumo 693-8501, Japan.ORCID 0000-0002-9280-8044
Shimane University · JPSecond Affiliated Hospital of Guangzhou Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Ataxia and Male Sterility (AMS) is a mutant mouse strain that contains a missense mutation in the coding region of

Indexed as

AnimalsAtaxiaFemaleGlutamineHeredodegenerative Disorders, Nervous SystemInfertility, MaleMaleMiceMice, Mutant StrainsNAD(P)H Dehydrogenase (Quinone)Neural Stem CellsTubulinGlutamineNAD(P)H Dehydrogenase (Quinone)Nqo1 protein, mouseTubulinAMS mousedeglutamylationMAP2NNA1NSCβ-tubulin

Identifiers

PMID33466875
PMCPMC7830091
OpenAlexW3120722209

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.