Evidence map›Paper›PMID 33425974›Full record

ReviewFrontiers in nutrition2020

Can the FUT2

Ottavia Giampaoli, Giorgia Conta, Riccardo Calvani, Alfredo Miccheli

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in nutrition, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
3.1field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 28 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Article
  5. Review
  6. Article
  7. Article
  8. Review
  9. Article
  10. Article
  11. Article
  12. Article
  13. Host gene effects on gut microbiota in type 1 diabetes.Biochemical Society transactions · 2022
    Review
  14. Gastrointestinal microbiome andWorld journal of gastroenterology · 2022
    Article
  15. FUT2 gene as a genetic susceptible marker of infectious diseases: A Review.International journal of molecular epidemiology and genetics · 2022
    Review
  16. Article
  17. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Ottavia GiampaoliDepartment of Chemistry, Sapienza University of Rome, Rome, Italy.
Giorgia ContaDepartment of Chemistry, Sapienza University of Rome, Rome, Italy.
Riccardo CalvaniFondazione Policlinico Universitario 'Agostino Gemelli' IRCCS, Rome, Italy.
Alfredo MiccheliNMR-Based Metabolomics Laboratory (NMLab), Sapienza University of Rome, Rome, Italy.
Sapienza University of Rome · ITAgostino Gemelli University Polyclinic · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The global toll of type 1 diabetes (T1D) has steadily increased over the last decades. It is now widely acknowledged that T1D pathophysiology is more complex than expected. Indeed, a multifaceted interplay between genetic, metabolic, inflammatory and environmental factors exists that leads to heterogeneous clinical manifestations across individuals. Children with

Indexed as

bifidobacteriaFUT2 genegut microbiotaHMOsnon-secretorsecretorshort-chain fatty acids (SCFA)T1D

Identifiers

PMID33425974
PMCPMC7785815
OpenAlexW3116242225

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.