Evidence map›Paper›PMID 33417723›Full record

ArticleMicrocirculation (New York, N.Y. : 1994)2021

Distinct structural and functional angiogenic responses are induced by different mechanical stimuli.

Roger W P Kissane, Peter G Tickle, Natalie E Doody, Abdullah A Al-Shammari, Stuart Egginton

Open access · hybridAbstract readEvaluation Study
In one paragraph

Article in Microcirculation (New York, N.Y. : 1994), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
1.0field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 10 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 2 countries.

Roger W P KissaneDepartment of Musculoskeletal & Ageing Science, University of Liverpool, Liverpool, UK.ORCID 0000-0001-9385-2584
Peter G TickleSchool of Biomedical Sciences, University of Leeds, Leeds, UK.
Natalie E DoodySchool of Biomedical Sciences, University of Leeds, Leeds, UK.
Abdullah A Al-ShammariDepartment of Mathematics, Faculty of Sciences, Kuwait University, Khaldiya, Kuwait.ORCID 0000-0002-4512-1151
Stuart EggintonSchool of Biomedical Sciences, University of Leeds, Leeds, UK.ORCID 0000-0002-3084-9692
University of Leeds · GBKuwait University · KW

Funding

British Heart Foundation PG/14/15/30691
6 · The paper itself

Abstract

objectiveAdequacy of the microcirculation is essential for maintaining repetitive skeletal muscle function while avoiding fatigue. It is unclear, however, whether capillary remodelling after different angiogenic stimuli is comparable in terms of vessel distribution and consequent functional adaptations. We determined the physiological consequences of two distinct mechanotransductive stimuli: (1) overload-mediated abluminal stretch (OV); (2) vasodilator-induced shear stress (prazosin, PR).

methodsIn situ EDL fatigue resistance was determined after 7 or 14 days of intervention, in addition to measurements of femoral artery flow. Microvascular composition (muscle histology) and oxidative capacity (citrate synthase activity) were quantified, and muscle PO

resultsCompared to controls, capillary-to-fiber ratio was higher after OV14 (134%, p < .001) and PR14 (121%, p < .05), although fatigue resistance only improved after overload (7 days: 135%, 14 days: 125%, p < .05). In addition, muscle overload improved local capillary supply indices and reduced CS activity, while prazosin treatment failed to alter either index of aerobic capacity.

conclusionTargeted capillary growth in response to abluminal stretch is a potent driver of improved muscle fatigue resistance, while shear stress-driven angiogenesis has no beneficial effect on muscle function. In terms of capillarity, more is not necessarily better.

Indexed as

Adrenergic alpha-1 Receptor AntagonistsCapillariesMotor ActivityMuscle, SkeletalNeovascularization, PhysiologicPrazosinAnimalsBiomechanical PhenomenaElectric StimulationMaleMicrocirculationMicrovesselsModels, AnimalMuscle FatigueOxygen ConsumptionRatsAdrenergic alpha-1 Receptor AntagonistsPrazosincapillary supplymathematical modellingoverloadshear stressskeletal muscle

Identifiers

PMID33417723
PMCPMC8614118
OpenAlexW3120862156

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.