Evidence map›Paper›PMID 33408641›Full record

ArticleFrontiers in physiology2020

The Role of Orai1 in Regulating Sarcoplasmic Calcium Release, Mitochondrial Morphology and Function in Myostatin Deficient Skeletal Muscle.

Mónika Sztretye, Zoltán Singlár, Norbert Balogh, Gréta Kis, Péter Szentesi, Ágnes Angyal, Ildikó Balatoni, László Csernoch, Beatrix Dienes

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Frontiers in physiology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
0.3field-weighted citation impact, top 44% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 6 citations in OpenAlex.

  1. Loss of Calpain 3 dysregulates store-operated calcium entry and its exercise response in mice.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2024
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 1 institution in 1 country.

Mónika SztretyeDepartment of Physiology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Zoltán SinglárDepartment of Physiology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Norbert BaloghDepartment of Physiology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Gréta KisDepartment of Anatomy, Histology and Embryology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Péter SzentesiDepartment of Physiology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Ágnes AngyalDepartment of Physiology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Ildikó BalatoniDepartment of Physiology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
László CsernochDepartment of Physiology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Beatrix DienesDepartment of Physiology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
University of Debrecen · HU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In mice a naturally occurring 12-bp deletion in the myostatin gene is considered responsible for the compact phenotype (Mstn

Indexed as

excitation contraction couplingmithochondrial defectmitochondrial calcium uptakemyostatin deficiencySOCE

Identifiers

PMID33408641
PMCPMC7779810
OpenAlexW3117497144

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.