ArticleEcology and evolution2020
Numbers, neighbors, and hungry predators: What makes chemically defended aposematic prey susceptible to predation?
Article in Ecology and evolution, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
4 citing papers in PubMed, 7 citations in OpenAlex.
- Don't You Know That I'm Toxic? Wild Birds Learn to Avoid a Novel Aposematic Warning Signal.Ecology and evolution · 2025Article
- Outstanding issues in the study of antipredator defenses.Ecology and evolution · 2023Review
- Increasingly cautious sampling, not the black colouration of unpalatable prey, is used by fish in avoidance learning.Animal cognition · 2023Article
- Numbers, neighbors, and hungry predators: What makes chemically defended aposematic prey susceptible to predation?Ecology and evolution · 2020Article
Corrections and comments
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Authors and funding
4 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Many chemically defended aposematic species are characterized by relatively low toxin levels, which enables predators to include them in their diets under certain circumstances. Knowledge of the conditions governing the survival of such prey animals-especially in the context of the co-occurrence of similar but undefended prey, which may result in mimicry-like interactions-is crucial for understanding the initial evolution of aposematism. In a one-month outdoor experiment using fish (the common carp
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.