Evidence map›Paper›PMID 33379346›Full record

ArticleInternational journal of molecular sciences2020

Inhibitory Effect of (2

Eun-Nam Kim, Ga-Ram Kim, Jae Sik Yu, Ki Hyun Kim, Gil-Saeng Jeong

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
0.6field-weighted citation impact, top 34% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 12 citations in OpenAlex.

  1. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Eun-Nam KimCollege of Pharmacy, Keimyung University, Daegu 42601, Korea.
Ga-Ram KimCollege of Pharmacy, Keimyung University, Daegu 42601, Korea.
Jae Sik YuSchool of Pharmacy, Sungkyunkwan University, Suwon 16419, Korea.
Ki Hyun KimSchool of Pharmacy, Sungkyunkwan University, Suwon 16419, Korea.ORCID 0000-0002-5285-9138
Gil-Saeng JeongCollege of Pharmacy, Keimyung University, Daegu 42601, Korea.
Keimyung University · KRSungkyunkwan University · KR

Funding

National Research Foundation of Korea 2019R1A5A2027340
6 · The paper itself

Abstract

In bone homeostasis, bone loss due to excessive osteoclasts and inflammation or osteolysis in the bone formation process cause bone diseases such as osteoporosis. Suppressing the accompanying oxidative stress such as ROS in this process is an important treatment strategy for bone disease. Therefore, in this study, the effect of (2

Indexed as

Cell DifferentiationAnimalsAntioxidantsButanolsCell DeathGene Expression RegulationLipopolysaccharidesMacrophagesMAP Kinase Signaling SystemMiceNF-kappa BOsteoblastsOsteoclastsRANK LigandRAW 264.7 CellsReactive Oxygen Species4-(4-hydroxyphenyl)-2-butanolAntioxidantsButanolsLipopolysaccharidesNF-kappa BRANK LigandReactive Oxygen SpeciesBetula platyphylla var. japonicacatalase (CAT)osteoclastreactive oxygen species (ROS)superoxide dismutase (SOD)

Identifiers

PMID33379346
PMCPMC7795186
OpenAlexW3114183578

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.