Evidence map›Paper›PMID 33361757›Full record

ArticleSignal transduction and targeted therapy2020

Kindlin-2 regulates skeletal homeostasis by modulating PTH1R in mice.

Xuekun Fu, Bo Zhou, Qinnan Yan, Chu Tao, Lei Qin, Xiaohao Wu, Sixiong Lin, Sheng Chen, Yumei Lai, Xuenong Zou and 8 more

Open access · goldAbstract read
In one paragraph

Article in Signal transduction and targeted therapy, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 42 papers.

0numbers the graph read from it
0cells of the map it votes in
42citing papers in PubMed
4.4field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

42 citing papers in PubMed, 55 citations in OpenAlex.

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  12. The p53-miR17 family-Rankl axis bridges liver-bone communication.Molecular therapy : the journal of the American Society of Gene Therapy · 2025
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

18 authors at 8 institutions in 3 countries.

Xuekun FuSchool of Medicine, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, and Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, 1088 Xue Yuan Road, 518055, Shenzhen, Guangdong, China.
Bo ZhouDepartment of Biology, Southern University of Science and Technology, 518055, Shenzhen, China.ORCID 0000-0003-4213-726X
Qinnan YanSchool of Medicine, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, and Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, 1088 Xue Yuan Road, 518055, Shenzhen, Guangdong, China.
Chu TaoSchool of Medicine, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, and Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, 1088 Xue Yuan Road, 518055, Shenzhen, Guangdong, China.
Lei QinSchool of Medicine, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, and Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, 1088 Xue Yuan Road, 518055, Shenzhen, Guangdong, China.
Xiaohao WuSchool of Medicine, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, and Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, 1088 Xue Yuan Road, 518055, Shenzhen, Guangdong, China.
Sixiong LinSchool of Medicine, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, and Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, 1088 Xue Yuan Road, 518055, Shenzhen, Guangdong, China.ORCID 0000-0001-7155-5044
Sheng ChenSchool of Medicine, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, and Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, 1088 Xue Yuan Road, 518055, Shenzhen, Guangdong, China.
Yumei LaiDepartment of Orthopedic Surgery, Rush University Medical Center, Chicago, IL, 60612, USA.
Xuenong ZouDepartment of Spine Surgery, Orthopedic Research Institute, The First Affiliated Hospital of Sun Yat-sen University, Guangdong Provincial Key Laboratory of Orthopedics and Traumatology, 510080, Guangzhou, China.
Zengwu ShaoDepartment of Orthopedics, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, 430022, Wuhan, China.
Meiqing WangState Key Laboratory of Military Stomatology, National Clinical Research Center for Oral Diseases, Shanxi International Joint Research Center for Oral Diseases, Department of Oral Anatomy and Physiology and TMD, School of Stomatology, the Fourth Military Medical University, 145 Changle West Road, Xi'an, China.
Di ChenResearch Center for Human Tissues and Organs Degeneration, Shenzhen Institutes of Advanced Technology, Chinese Academy of Sciences, 518055, Shenzhen, China.
Wenfei JinDepartment of Biology, Southern University of Science and Technology, 518055, Shenzhen, China.ORCID 0000-0002-0028-3739
Youqiang SongSchool of Biomedical Sciences, University of Hong Kong, 21 Sassoon Road, Hong Kong, China.
Huiling CaoSchool of Medicine, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, and Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, 1088 Xue Yuan Road, 518055, Shenzhen, Guangdong, China. caohl@sustech.edu.cn.
Ge ZhangLaw Sau Fai Institute for Advancing Translational Medicine in Bone and Joint Diseases, School of Chinese Medicine, Hong Kong Baptist University, 999077, Hong Kong, SAR, China. zhangge@hkbu.edu.hk.
Guozhi XiaoSchool of Medicine, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, and Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, 1088 Xue Yuan Road, 518055, Shenzhen, Guangdong, China. xiaogz@sustech.edu.cn.ORCID 0000-0002-4269-2450
Southern University of Science and Technology · CNHong Kong Baptist University · HKSun Yat-sen University · CNUnion Hospital · CNAir Force Medical University · CNChinese Academy of Sciences · CNRush University Medical Center · USUniversity of Hong Kong · HK

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In vertebrates, the type 1 parathyroid hormone receptor (PTH1R) is a critical regulator of skeletal development and homeostasis; however, how it is modulated is incompletely understood. Here we report that deleting Kindlin-2 in osteoblastic cells using the mouse 10-kb Dmp1-Cre largely neutralizes the intermittent PTH-stimulated increasing of bone volume fraction and bone mineral density by impairing both osteoblast and osteoclast formation in murine adult bone. Single-cell profiling reveals that Kindlin-2 loss increases the proportion of osteoblasts, but not mesenchymal stem cells, chondrocytes and fibroblasts, in non-hematopoietic bone marrow cells, with concomitant depletion of osteoblasts on the bone surfaces, especially those stimulated by PTH. Furthermore, haploinsufficiency of Kindlin-2 and Pth1r genes, but not that of either gene, in mice significantly decreases basal and, to a larger extent, PTH-stimulated bone mass, supporting the notion that both factors function in the same genetic pathway. Mechanistically, Kindlin-2 interacts with the C-terminal cytoplasmic domain of PTH1R via aa 474-475 and Gsα. Kindlin-2 loss suppresses PTH induction of cAMP production and CREB phosphorylation in cultured osteoblasts and in bone. Interestingly, PTH promotes Kindlin-2 expression in vitro and in vivo, thus creating a positive feedback regulatory loop. Finally, estrogen deficiency induced by ovariectomy drastically decreases expression of Kindlin-2 protein in osteocytes embedded in the bone matrix and Kindlin-2 loss essentially abolishes the PTH anabolic activity in bone in ovariectomized mice. Thus, we demonstrate that Kindlin-2 functions as an intrinsic component of the PTH1R signaling pathway in osteoblastic cells to regulate bone mass accrual and homeostasis.

Indexed as

HomeostasisAnimalsBone and BonesCytoskeletal ProteinsMiceMice, TransgenicMuscle ProteinsOsteoblastsReceptor, Parathyroid Hormone, Type 1Cytoskeletal Proteinskindlin-2 protein, mouseMuscle ProteinsPTH1R protein, mouseReceptor, Parathyroid Hormone, Type 1

Identifiers

PMID33361757
PMCPMC7762753
OpenAlexW3115531271

What OpenQuestion holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.