Evidence map›Paper›PMID 33361674›Full record

ReviewInternal medicine (Tokyo, Japan)2021

Hereditary Hemorrhagic Telangiectasia Induced Portosystemic Encephalopathy: A Case Report and Literature Review.

Hiroyuki Kawabata, Yasuhiko Hamada, Aiji Hattori, Kyosuke Tanaka

Open access · diamondAbstract readCase ReportsReview
In one paragraph

Review in Internal medicine (Tokyo, Japan), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
0.4field-weighted citation impact, top 36% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 3 citations in OpenAlex.

  1. Review
  2. Osler-Weber-Rendu Disease: Unexpected Cause of Anemia.Internal medicine (Tokyo, Japan) · 2023
    Article
  3. [A case of hepatic encephalopathy induced by hereditary hemorrhagic telangiectasia].Zhonghua gan zang bing za zhi = Zhonghua ganzangbing zazhi = Chinese journal of hepatology · 2022
    Article
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Hiroyuki KawabataDepartment of Gastroenterology, Saiseikai Matsusaka General Hospital, Japan.
Yasuhiko HamadaDepartment of Gastroenterology and Hepatology, Mie University Hospital, Japan.
Aiji HattoriDepartment of Gastroenterology, Saiseikai Matsusaka General Hospital, Japan.
Kyosuke TanakaDepartment of Endoscopy, Mie University Hospital, Japan.
Mie University Hospital · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hereditary hemorrhagic telangiectasia (HHT) is a rare disorder characterized by telangiectasias and arteriovenous malformations (AVMs), which can involve multiple organ systems. Although hepatic involvement is common, the development of portosystemic encephalopathy is extremely rare. We herein report a 72-year-old woman with HHT-induced portosystemic encephalopathy secondary to hepatic arteriovenous malformations. She presented with disturbance of consciousness, and her serum ammonia level was elevated at 270 mg/dL. Color Doppler ultrasonography and contrast-enhanced computed tomography showed hepatic AVMs and shunts, which were useful for making the definite diagnosis. Portosystemic encephalopathy should be considered as a differential diagnosis in HHT patients presenting with disturbance of consciousness.

Indexed as

Arteriovenous MalformationsHepatic EncephalopathyLiver DiseasesTelangiectasia, Hereditary HemorrhagicAgedFemaleHumansTomography, X-Ray Computedarteriovenous malformationhereditary hemorrhagic telangiectasiaportosystemic encephalopathy

Identifiers

PMID33361674
PMCPMC8188034
OpenAlexW3115377804

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.