Evidence map›Paper›PMID 33294263›Full record

ArticleAmerican journal of cancer research2020

ERα is a target for butein-induced growth suppression in breast cancer.

Chuyi Huang, Xiaohong Xia, Jinchan He, Yuan Liu, Zhenlong Shao, Tumei Hu, Cuifu Yu, Xiaolin Liu, Qiong Xu, Bin Liu and 3 more

Open access · greenAbstract read
In one paragraph

Article in American journal of cancer research, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
1.1field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 7 citations in OpenAlex.

  1. Article
  2. Article
  3. A SIX1 degradation inducer blocks excessive proliferation of prostate cancer.International journal of biological sciences · 2022
    Article
  4. Review
  5. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 2 institutions in 1 country.

Chuyi HuangAffiliated Cancer Hospital & Institute of Guangzhou Medical University Guangzhou 510095, Guangdong, China.
Xiaohong XiaAffiliated Cancer Hospital & Institute of Guangzhou Medical University Guangzhou 510095, Guangdong, China.
Jinchan HeAffiliated Cancer Hospital & Institute of Guangzhou Medical University Guangzhou 510095, Guangdong, China.
Yuan LiuAffiliated Cancer Hospital & Institute of Guangzhou Medical University Guangzhou 510095, Guangdong, China.
Zhenlong ShaoAffiliated Cancer Hospital & Institute of Guangzhou Medical University Guangzhou 510095, Guangdong, China.
Tumei HuAffiliated Cancer Hospital & Institute of Guangzhou Medical University Guangzhou 510095, Guangdong, China.
Cuifu YuAffiliated Cancer Hospital & Institute of Guangzhou Medical University Guangzhou 510095, Guangdong, China.
Xiaolin LiuGuangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University Guangzhou 510260, Guangdong, China.
Qiong XuGuangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University Guangzhou 510260, Guangdong, China.
Bin LiuGuangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University Guangzhou 510260, Guangdong, China.
Ningning LiuGuangzhou Institute of Cardiovascular Disease, Guangdong Key Laboratory of Vascular Diseases, State Key Laboratory of Respiratory Disease, The Second Affiliated Hospital, Guangzhou Medical University Guangzhou 510260, Guangdong, China.
Yuning LiaoAffiliated Cancer Hospital & Institute of Guangzhou Medical University Guangzhou 510095, Guangdong, China.
Hongbiao HuangAffiliated Cancer Hospital & Institute of Guangzhou Medical University Guangzhou 510095, Guangdong, China.
Guangzhou Medical University Cancer Hospital · CNState Key Laboratory of Respiratory Disease · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Breast cancer (BCa) has the highest incidence and mortality among malignant diseases in female worldwide. BCa is frequently caused by estrogen receptor α (ERα), a ligand-dependent receptor that highly expressed in about 70% of breast tumors. Therefore, ERα has become a well-characterized and the most effective target for treating ERα-expressing BCa (ERα

Indexed as

Breast cancerbuteindegradationestrogen receptor αgrowth

Identifiers

PMID33294263
PMCPMC7716169
OpenAlexW3112187253

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.