Article in Cells, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.2field-weighted citation impact, top 49% of its field
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literature
Who cites it
1 citing paper in PubMed, 2 citations in OpenAlex.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
9 authors at 4 institutions in 3 countries.
David S GutteryQueens Medical Centre, School of Life Sciences, University of Nottingham, Nottingham NG7 2UH, UK.ORCID 0000-0003-0418-580X
Abhinay RamaprasadBiological and Environmental Sciences and Engineering (BESE) Division, King Abdullah University of Science and Technology, Thuwal 23955, Saudi Arabia.
David J P FergusonNuffield Department of Clinical Laboratory Science, University of Oxford, John Radcliffe Hospital, Oxford OX1 2JD, UK.ORCID 0000-0001-5045-819X
Mohammad ZeeshanQueens Medical Centre, School of Life Sciences, University of Nottingham, Nottingham NG7 2UH, UK.
Rajan PandeyQueens Medical Centre, School of Life Sciences, University of Nottingham, Nottingham NG7 2UH, UK.
Declan BradyQueens Medical Centre, School of Life Sciences, University of Nottingham, Nottingham NG7 2UH, UK.ORCID 0000-0001-9150-7840
Anthony A HolderThe Francis Crick Institute, London NW1 1AT, UK.
Arnab PainBiological and Environmental Sciences and Engineering (BESE) Division, King Abdullah University of Science and Technology, Thuwal 23955, Saudi Arabia.
Rita TewariQueens Medical Centre, School of Life Sciences, University of Nottingham, Nottingham NG7 2UH, UK.
University of Nottingham · GBThe Francis Crick Institute · GBHokkaido University · JPOxford Brookes University · GB
Funding
Cancer Research UK FC010097Medical Research Council FC010097Medical Research Council G0900109Medical Research Council G0900278Medical Research Council MR/K011782/1Wellcome Trust FC010097
6 · The paper itself
Abstract
The meiotic recombination 11 protein (MRE11) plays a key role in DNA damage response and maintenance of genome stability. However, little is known about its function during development of the malaria parasite
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
MRE11 Is Crucial for Malaria Parasite Transmission and Its Absence Affects Expression of Interconnected Networks of Key Genes Essential for Life. · full record | OpenQuestion