Evidence map›Paper›PMID 33206363›Full record

ArticleMolecular biology reports2020

Netrin-1 protects the SH-SY5Y cells against amyloid beta neurotoxicity through NF-κB/Nrf2 dependent mechanism.

Elham Zamani, Mohsen Parviz, Mehrdad Roghani, Marjan Hosseini, Parvaneh Mohseni-Moghaddam, Marjan Nikbakhtzadeh

Abstract read
PubMed Publisher
In one paragraph

Article in Molecular biology reports, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
0.8field-weighted citation impact, top 32% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 19 citations in OpenAlex.

  1. Photolytic disruption of Alzheimer's amyloid AβCommunications chemistry · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 1 country.

Elham ZamaniElectrophysiology Research Center, Neuroscience Institute, Tehran University of Medical Sciences, Tehran, Iran.
Mohsen ParvizElectrophysiology Research Center, Neuroscience Institute, Tehran University of Medical Sciences, Tehran, Iran. parvizmo@tums.ac.ir.ORCID http://orcid.org/0000-0002-2988-3551
Mehrdad RoghaniNeurophysiology Research Center, Shahed University, Tehran, Iran.
Marjan HosseiniDepartment of Physiology, School of Medicine, Tehran University of Medical Sciences, Pour Sina Street, Keshavarz Boulevard, Tehran, Iran.
Parvaneh Mohseni-MoghaddamDepartment of Physiology, School of Medicine, Tehran University of Medical Sciences, Pour Sina Street, Keshavarz Boulevard, Tehran, Iran.
Marjan NikbakhtzadehDepartment of Physiology, School of Medicine, Tehran University of Medical Sciences, Pour Sina Street, Keshavarz Boulevard, Tehran, Iran.
Tehran University of Medical Sciences · IRShahed University · IRSina Hospital · IR

Funding

Electrophysiology Research Center, Neuroscience Institute, Tehran University of Medical Sciences 33292-139-04-95
6 · The paper itself

Abstract

Many evidence confirms that amyloid beta 1-42 fragment (Aβ

Indexed as

Alzheimer DiseaseAmyloid beta-PeptidesApoptosisCell Line, TumorCell SurvivalHumansNetrin-1NeuroblastomaNF-E2-Related Factor 2NF-kappa BOxidative StressPeptide FragmentsProtective AgentsReactive Oxygen SpeciesRecombinant ProteinsSignal TransductionAmyloid beta-Peptidesamyloid beta-protein (1-42)Netrin-1NFE2L2 protein, humanNF-E2-Related Factor 2NF-kappa BNTN1 protein, humanPeptide FragmentsProtective AgentsReactive Oxygen SpeciesRecombinant ProteinsTNF protein, humanTumor Necrosis Factor-alphaAlzheimer's diseaseAmyloid beta 1–42InflammationNetrin-1SH-SY5Y cell line

Identifiers

PMID33206363
OpenAlexW3098034806

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.