Evidence map›Paper›PMID 33198056›Full record

ArticleCells2020

Autophagy Activated by Peroxiredoxin of

Xia Li, Yuhan Zhang, Yanqing Zhao, Ke Qiao, Meng Feng, Hang Zhou, Hiroshi Tachibana, Xunjia Cheng

Open access · goldAbstract read
In one paragraph

Article in Cells, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
0.2field-weighted citation impact, top 38% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 8 citations in OpenAlex.

  1. Interactions ofMicrobiology spectrum · 2026
    Article
  2. The role of autophagy in protozoan infections.Frontiers in cellular and infection microbiology · 2026
    Review
  3. Article
  4. Article
  5. Article
  6. Article
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 2 countries.

Xia LiDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Fudan University, Shanghai 200000, China.
Yuhan ZhangDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Fudan University, Shanghai 200000, China.
Yanqing ZhaoDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Fudan University, Shanghai 200000, China.
Ke QiaoInstitute of Metabolic & Integrative Biology, Fudan University, Shanghai 200438, China.
Meng FengDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Fudan University, Shanghai 200000, China.
Hang ZhouDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Fudan University, Shanghai 200000, China.
Hiroshi TachibanaDepartment of Infectious Diseases, Tokai University School of Medicine, Isehara, Kanagawa 259-1193, Japan.
Xunjia ChengDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Fudan University, Shanghai 200000, China.ORCID 0000-0002-8851-6903
Shanghai Medical College of Fudan University · CNTokai University · JPFudan University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Autophagy, an evolutionarily conserved mechanism to remove redundant or dangerous cellular components, plays an important role in innate immunity and defense against pathogens, which, in turn, can regulate autophagy to establish infection within a host. However, for

Indexed as

AutophagyAdaptor Proteins, Vesicular TransportAnimalsAutophagosomesEntamoeba histolyticaFemaleMiceMice, Inbred C57BLPeroxiredoxinsProtein DomainsRAW 264.7 CellsSignal TransductionToll-Like Receptor 4Adaptor Proteins, Vesicular TransportPeroxiredoxinsTicam1 protein, mouseToll-Like Receptor 4autophagyEntamoeba histolyticainnate immunitymacrophageperoxiredoxintoll-like receptor 4

Identifiers

PMID33198056
PMCPMC7696310
OpenAlexW3098148461

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.