Evidence map›Paper›PMID 33143308›Full record

ArticleInternational journal of molecular sciences2020

SATMF Suppresses the Premature Senescence Phenotype of the ATM Loss-of-Function Mutant and Improves Its Fertility in

Yi Zhang, Hou-Ling Wang, Yuhan Gao, Hongwei Guo, Zhonghai Li

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.9field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 8 citations in OpenAlex.

  1. Article
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 3 institutions in 1 country.

Yi ZhangBeijing Advanced Innovation Center for Tree Breeding by Molecular Design, Beijing Forestry University, Beijing 100083, China.
Hou-Ling WangBeijing Advanced Innovation Center for Tree Breeding by Molecular Design, Beijing Forestry University, Beijing 100083, China.ORCID 0000-0002-9950-6866
Yuhan GaoKey Laboratory of Pest Management in Crops of the Ministry of Agriculture, Institute of Plant Protection, Chinese Academy of Agricultural Sciences, Beijing 100193, China.
Hongwei GuoBeijing Advanced Innovation Center for Tree Breeding by Molecular Design, Beijing Forestry University, Beijing 100083, China.
Zhonghai LiBeijing Advanced Innovation Center for Tree Breeding by Molecular Design, Beijing Forestry University, Beijing 100083, China.
Beijing Forestry University · CNInstitute of Plant Protection · CNSouthern University of Science and Technology · CN

Funding

Chinese Postdoctoral Science Foundation 2019M650514Chinese Postdoctoral Science Foundation 2019M650516Chinese Postdoctoral Science Foundation 2020M670544National Key Research and Development Program of China No. 2019YFA0903904National Natural Science Foundation of China 31570286National Natural Science Foundation of China 31900173National Natural Science Foundation of China 31970196Shenzhen Science and Technology Innovation Program No. KQTD20190929173906742
6 · The paper itself

Abstract

Leaf senescence is the final stage of leaf development. It is accompanied by the remobilization of nutrients from senescent leaves to developing organs. The occurrence of senescence is the consequence of integrating intrinsic and environmental signals. DNA damage triggered by stresses has been regarded as one of the reasons for senescence. To prevent DNA damage, cells have evolved elaborate DNA repair machinery. The ataxia telangiectasia mutated (ATM) functions as the chief transducer of the double-strand breaks (DSBs) signal. Our previous study suggests that ATM functions in lifespan regulation in

Indexed as

Cellular SenescenceFertilityArabidopsisArabidopsis ProteinsAtaxia Telangiectasia Mutated ProteinsHumansLoss of Function MutationPhenotypePlant LeavesArabidopsis ProteinsAtaxia Telangiectasia Mutated ProteinsATM protein, ArabidopsisATMDNA damagefertilityleaf senescenceSATMF

Identifiers

PMID33143308
PMCPMC7662627
OpenAlexW3097378726

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.