Evidence map›Paper›PMID 33139082›Full record

ReviewTrends in neurosciences2020

Alternative Frameworks for Advancing the Study of Eating Disorders.

Sarah A Stern, Cynthia M Bulik

Open access · hybridAbstract readReview
In one paragraph

Review in Trends in neurosciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
0.7field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 20 citations in OpenAlex.

  1. Article
  2. Identification of a neural basis for anorexia nervosa.bioRxiv : the preprint server for biology · 2026
    Article
  3. Article
  4. Article
  5. Perceptual Dysfunction in Eating Disorders.Current topics in behavioral neurosciences · 2025
    Review
  6. Review
  7. Article
  8. Article
  9. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 2 countries.

Sarah A SternMax Planck Florida Institute for Neuroscience, Jupiter, FL, USA; Department of Molecular Genetics, Rockefeller University, New York, NY, USA.
Cynthia M BulikDepartment of Psychiatry, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA; Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden; Department of Nutrition, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA. Electronic address: cbulik@med.unc.edu.
Max Planck Florida Institute for Neuroscience · USUniversity of North Carolina at Chapel Hill · US

Funding

Eating Disorders Genetics Initiative (EDGI)R01MH120170 · NIMH · UNIV OF NORTH CAROLINA CHAPEL HILL · PI BULIK, CYNTHIA M · 2019 to 2023
$7.2M
Microbiome-mediated weight, anxiety, and stress dysregulation in anorexia nervosaR01MH105684 · NIMH · UNIV OF NORTH CAROLINA CHAPEL HILL · PI CARROLL, IAN MICHAEL · 2015 to 2019
$3.9M
Predicting the multi-omic impact of psychiatric GWAS associationsR01MH118278 · NIMH · YALE UNIVERSITY · PI HUCKINS, LAURA MARIANNE · 2019 to 2023
$3.6M
Predicting Binge and Purge Episodes from Passive and Active Apple Watch Data Using a Dynamical Systems ApproachR01MH119084 · NIMH · UNIV OF NORTH CAROLINA CHAPEL HILL · PI BULIK, CYNTHIA M, BUTNER, JONATHAN · 2019 to 2022
$2.8M
1/7 Psychiatric Genomics Consortium: Finding actionable variationU01MH109528 · NIMH · UNIV OF NORTH CAROLINA CHAPEL HILL · PI SULLIVAN, PATRICK F · 2016 to 2020
$2.5M
Neural circuit mechanisms controlling non-homeostatic feedingR00DA048749 · NIDA · MAX PLANCK FLORIDA CORPORATION · PI STERN, SARAH · 2021 to 2023
$848k
Premorbid metabolism and growth in anorexia nervosa: A genetically informed exploration of riskR21MH115397 · NIMH · UNIV OF NORTH CAROLINA CHAPEL HILL · PI BULIK, CYNTHIA M, MICALI, NADIA · 2017 to 2018
$362k
Neural circuit mechanisms controlling non-homeostatic feedingK99DA048749 · NIDA · ROCKEFELLER UNIVERSITY · PI STERN, SARAH · 2020 to 2020
$163k
NIDA NIH HHS K99 DA048749NIDA NIH HHS R00 DA048749NIMH NIH HHS R01 MH105684NIMH NIH HHS R01 MH118278NIMH NIH HHS R01 MH119084NIMH NIH HHS R01 MH120170NIMH NIH HHS R21 MH115397NIMH NIH HHS U01 MH109528
6 · The paper itself

Abstract

Eating disorders are life-interrupting psychiatric conditions with high morbidity and mortality, yet the basic mechanisms underlying these conditions are understudied compared with other psychiatric disorders. In this opinion, we suggest that recent knowledge gleaned from genomic and neuroimaging investigations of eating disorders in humans presents a rich opportunity to sharpen animal models of eating disorders and to identify neural mechanisms that contribute to the risk and maintenance of these conditions. Our article reflects the state of the science, with a primary focus on anorexia nervosa (AN) and binge-eating behavior, and encourages further study of all conditions categorized under feeding and eating disorders.

Indexed as

Anorexia NervosaBinge-Eating DisorderFeeding and Eating DisordersAnimalsHumansModels, AnimalNeuroimaginganimal modelsanorexia nervosagenomicsGWASneural circuitsneuroimaging

Identifiers

PMID33139082
PMCPMC8627201
OpenAlexW3095569865

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.