Evidence map›Paper›PMID 33132828›Full record

ArticleFrontiers in neuroscience2020

Effects of Akt/mTOR/p70S6K Signaling Pathway Regulation on Neuron Remodeling Caused by Translocation Repair.

Yusong Yuan, Dongdong Li, Fei Yu, Xuejing Kang, Hailin Xu, Peixun Zhang

Open access · goldAbstract read
In one paragraph

Article in Frontiers in neuroscience, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed, 1 pooled it
0.6field-weighted citation impact, top 38% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 1 synthesis or guideline pooled it, 11 citations in OpenAlex.

  1. Pooled it
  2. Article
  3. Article
  4. Article
  5. International journal of molecular sciences · 2023
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Yusong YuanDepartment of Trauma and Orthopedics, Peking University People's Hospital, Peking University, Beijing, China.
Dongdong LiDepartment of Trauma and Orthopedics, Peking University People's Hospital, Peking University, Beijing, China.
Fei YuDepartment of Trauma and Orthopedics, Peking University People's Hospital, Peking University, Beijing, China.
Xuejing KangDepartment of Trauma and Orthopedics, Peking University People's Hospital, Peking University, Beijing, China.
Hailin XuDepartment of Trauma and Orthopedics, Peking University People's Hospital, Peking University, Beijing, China.
Peixun ZhangDepartment of Trauma and Orthopedics, Peking University People's Hospital, Peking University, Beijing, China.
Peking University People's Hospital · CNPeking University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Peripheral nerve injury repair has been considered a difficult problem in the field of trauma for a long time. Conventional surgical methods are not applicable in some special types of nerve injury, prompting scholars to seek to develop more effective nerve translocation repair technologies. The purpose of this study was to explore the functional state of neurons in injured lower limbs after translocation repair, with a view to preliminarily clarify the molecular mechanisms underlying this process. Eighteen Sprague-Dawley rats were divided into the normal, tibial nerve

Indexed as

dorsal root ganglionperipheral nerve injuryremodelingsignaling pathwaytranslocation repair

Identifiers

PMID33132828
PMCPMC7550644
OpenAlexW3091726493

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.