Evidence map›Paper›PMID 33102799›Full record

ReviewEndocrinology, diabetes & metabolism2020

Human and molecular genetics shed lights on fatty liver disease and diabetes conundrum.

Federica Tavaglione, Giovanni Targher, Luca Valenti, Stefano Romeo

Open access · goldAbstract readReview
In one paragraph

Review in Endocrinology, diabetes & metabolism, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
1.6field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 15 citations in OpenAlex.

  1. Article
  2. Observational
  3. Review
  4. Review
  5. Article
  6. Article
  7. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 4 institutions in 2 countries.

Federica TavaglioneClinical Medicine and Hepatology Unit Department of Internal Medicine and Geriatrics Campus Bio-Medico University Rome Italy.ORCID https://orcid.org/0000-0002-1720-4355
Giovanni TargherSection of Endocrinology, Diabetes and Metabolism Department of Medicine University and Azienda Ospedaliera Universitaria Integrata of Verona Verona Italy.
Luca ValentiDepartment of Pathophysiology and Transplantation Università degli Studi di Milano Milano Italy.
Stefano RomeoDepartment of Molecular and Clinical Medicine Sahlgrenska Academy University of Gothenburg Gothenburg Sweden.
Azienda Ospedaliera Universitaria Integrata Verona · ITSahlgrenska University Hospital · SEUniversità Campus Bio-Medico · ITUniversity of Milan · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The causal role of abdominal overweight/obesity, insulin resistance and type 2 diabetes (T2D) on the risk of fatty liver disease (FLD) has robustly been proven. A consensus of experts has recently proposed the novel definition of 'metabolic dysfunction-associated fatty liver disease, MAFLD' instead of 'nonalcoholic fatty liver disease, NAFLD', emphasizing the central role of dysmetabolism in the disease pathogenesis. Conversely, a direct and independent contribution of FLD

Indexed as

diabetesfatty liver diseasehuman genetics

Identifiers

PMID33102799
PMCPMC7576307
OpenAlexW3083695748

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.