Evidence map›Paper›PMID 33053858›Full record

ArticleJournal of clinical medicine2020

HbA1c and Aortic Calcification Index as Noninvasive Predictors of Pre-Existing Histopathological Damages in Living Donor Kidney Transplantation.

Kosuke Tanaka, Shigeyoshi Yamanaga, Yuji Hidaka, Sho Nishida, Kohei Kinoshita, Akari Kaba, Toshinori Ishizuka, Satoshi Hamanoue, Kenji Okumura, Chiaki Kawabata and 5 more

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Journal of clinical medicine, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.2field-weighted citation impact, top 40% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 2 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

15 authors at 2 institutions in 1 country.

Kosuke TanakaDepartment of Surgery, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Shigeyoshi YamanagaDepartment of Surgery, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Yuji HidakaDepartment of Surgery, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Sho NishidaDepartment of Surgery, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Kohei KinoshitaDepartment of Surgery, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Akari KabaDepartment of Surgery, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Toshinori IshizukaDepartment of Nephrology, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Satoshi HamanoueDepartment of Nephrology, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Kenji OkumuraDepartment of Surgery, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.ORCID 0000-0002-7751-2624
Chiaki KawabataDepartment of Nephrology, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Mariko ToyodaDepartment of Nephrology, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Asami TakedaDepartment of Nephrology, Japanese Red Cross Nagoya Daini Hospital, Aichi, 466-8650, Japan.
Akira MiyataDepartment of Nephrology, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Masayuki KashimaDepartment of Nephrology, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Hiroshi YokomizoDepartment of Surgery, Japanese Red Cross Kumamoto Hospital, Kumamoto, 861-8520, Japan.
Japanese Red Cross Kumamoto Hospital · JPJapanese Red Cross Nagoya Daini Hospital · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

We previously reported that allografts from living donors may have pre-existing histopathological damages, defined as the combination of interstitial fibrosis (ci), tubular atrophy (ct), and arteriolar hyalinosis (ah) scores of ≧1, according to the Banff classification. We examined preoperative characteristics to identify whether the degree of these damages was related to metabolic syndrome-related factors of donors. We conducted a single-center cross-sectional analysis including 183 living kidney donors. Donors were divided into two groups: chronic change (ci + ct ≧ 1 ∩ ah ≧ 1,

Indexed as

aortic calcification indexHbA1crenal agingrenal function recovery

Identifiers

PMID33053858
PMCPMC7601662
OpenAlexW3092307997

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.