Evidence map›Paper›PMID 33036460›Full record

ArticleInternational journal of molecular sciences2020

CX3CR1 Deficiency Attenuates DNFB-Induced Contact Hypersensitivity Through Skewed Polarization Towards M2 Phenotype in Macrophages.

Sayaka Otobe, Teruyoshi Hisamoto, Tomomitsu Miyagaki, Sohshi Morimura, Hiraku Suga, Makoto Sugaya, Shinichi Sato

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.5field-weighted citation impact, top 37% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 13 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Role of Innate Immunity in Allergic Contact Dermatitis: An Update.International journal of molecular sciences · 2023
    Review
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Sayaka OtobeDepartment of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
Teruyoshi HisamotoDepartment of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
Tomomitsu MiyagakiDepartment of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.ORCID 0000-0003-1879-3128
Sohshi MorimuraDepartment of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
Hiraku SugaDepartment of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
Makoto SugayaDepartment of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
Shinichi SatoDepartment of Dermatology, the University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan.
The University of Tokyo · JPSt. Marianna University School of Medicine · JP

Funding

the Ministry of Education, Culture, Sports, Science and Technology in Japan 16K19709the Ministry of Education, Culture, Sports, Science and Technology in Japan 26893052
6 · The paper itself

Abstract

CX3CL1 can function as both an adhesion molecule and a chemokine for CX3CR1

Indexed as

AnimalsBiomarkersCX3C Chemokine Receptor 1Dermatitis, ContactDinitrofluorobenzeneDisease Models, AnimalDisease SusceptibilityImmunohistochemistryMacrophage ActivationMacrophagesMiceMice, KnockoutNeutrophil InfiltrationBiomarkersCX3C Chemokine Receptor 1Dinitrofluorobenzenearginase-1contact hypersensitivityCX3CR1interleukin-6macrophagetumor necrosis factor-α

Identifiers

PMID33036460
PMCPMC7582565
OpenAlexW3092065342

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.