ArticlePostepy dermatologii i alergologii2020
The role of pentraxin 3 in pemphigus vulgaris.
Article in Postepy dermatologii i alergologii, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed, 1 citations in OpenAlex.
- Apoptosis and PTX3 in lichen planus.Postepy dermatologii i alergologii · 2025Article
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
introductionPemphigus is a group of autoimmune bullous diseases caused by antibodies directed against the desmosomal adhesion molecules desmoglein 1 and 3, which are required for intercellular adhesion of keratinocytes. Pentraxins are a group of proteins that function as pattern recognition molecules and also play a role in humoral innate immunity. Pentraxin 3 (PTX3) is the prototype of the long pentraxins and has been shown to be increased in numerous autoimmune diseases.
aimTo investigate whether PTX3 can be used as a marker of PV caused by autoimmunity and resulting in tissue injury. MATERIAL AND
methodsThe study included 30 patients who presented to the University Medical School Dermatology Department and were diagnosed with PV based on clinical, histological, and immunological findings. The control group included 30 healthy individuals. Human PTX3 concentration was measured with a commercially available ELISA kit, using a double antibody sandwich enzyme-linked immunosorbent assay.
resultsThe 60 participants comprised 31 (52%) men and 29 (48%) women. The most common site of onset was mucosa + skin (
conclusionsPTX3 was found to be increased in PV and PTX3 could be a useful indicator of disease activity in PV.
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