Evidence map›Paper›PMID 32924936›Full record

ArticleeLife2020

The many roles of C1q.

Mark Noble, Christoph Pröschel

Abstract readComment
In one paragraph

Article in eLife, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed.

  1. Article
  2. Article
  3. Review
  4. Article
  5. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

2 authors.

Mark NobleDepartment of Biomedical Genetics, University of Rochester School of Medicine and Dentistry, Rochester, United States.ORCID 0000-0002-2375-7773
Christoph PröschelDepartment of Biomedical Genetics, University of Rochester School of Medicine and Dentistry, Rochester, United States.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The ability of a well-known component of the complement cascade to bind to a variety of receptors has implications for signaling biology, spinal cord injury and, possibly, the evolution of the complement system.

Indexed as

Complement C1qNeural Stem CellsMembrane GlycoproteinsReceptors, Complementcomplement 1q receptorComplement C1qMembrane GlycoproteinsReceptors, Complementcomplement C1qmouseneural stem cell transplantneuroepithelial stem cellsregenerative medicinespinal cord injurystem cells

Identifiers

PMID32924936
PMCPMC7490008

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.