Evidence map›Paper›PMID 32915316›Full record

ArticlePflugers Archiv : European journal of physiology2020

Interleukin-1β in hypothalamic paraventricular nucleus mediates excitatory renal reflex.

Fen Zheng, Chao Ye, Guo-Wei Wan, Bing Zhou, Ying Tong, Jian-Zhen Lei, Qi Chen, Yue-Hua Li, Yu-Ming Kang, Guo-Qing Zhu

Abstract read
PubMed Publisher
In one paragraph

Article in Pflugers Archiv : European journal of physiology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
0.7field-weighted citation impact, top 36% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 11 citations in OpenAlex.

  1. Review
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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

10 authors at 3 institutions in 1 country.

Fen ZhengKey Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center of Translational Medicine for Cardiovascular Disease, and Department of Physiology, Nanjing Medical University, Nanjing, 211166, Jiangsu, China.
Chao YeKey Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center of Translational Medicine for Cardiovascular Disease, and Department of Physiology, Nanjing Medical University, Nanjing, 211166, Jiangsu, China.
Guo-Wei WanKey Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center of Translational Medicine for Cardiovascular Disease, and Department of Physiology, Nanjing Medical University, Nanjing, 211166, Jiangsu, China.
Bing ZhouKey Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center of Translational Medicine for Cardiovascular Disease, and Department of Physiology, Nanjing Medical University, Nanjing, 211166, Jiangsu, China.
Ying TongKey Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center of Translational Medicine for Cardiovascular Disease, and Department of Physiology, Nanjing Medical University, Nanjing, 211166, Jiangsu, China.
Jian-Zhen LeiKey Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center of Translational Medicine for Cardiovascular Disease, and Department of Physiology, Nanjing Medical University, Nanjing, 211166, Jiangsu, China.
Qi ChenDepartment of Pathophysiology, Nanjing Medical University, Nanjing, 211166, Jiangsu, China.
Yue-Hua LiDepartment of Pathophysiology, Nanjing Medical University, Nanjing, 211166, Jiangsu, China.
Yu-Ming KangDepartment of Physiology and Pathophysiology, Cardiovascular Research Center, Xi'an Jiaotong University School of Medicine, Xi'an, 710061, China.
Guo-Qing ZhuKey Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center of Translational Medicine for Cardiovascular Disease, and Department of Physiology, Nanjing Medical University, Nanjing, 211166, Jiangsu, China. gqzhucn@njmu.edu.cn.ORCID 0000-0002-3132-9592
Nanjing Medical University · CNNanjing University of Chinese Medicine · CNXi'an Jiaotong University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Chemical stimulation of kidney causes sympathetic activation and pressor responses in rats. The excitatory renal reflex (ERR) is mediated by angiotensin type 1 receptor (AT

Indexed as

ReflexAcetophenonesAngiotensin II Type 1 Receptor BlockersAnimalsBlood PressureCapsaicinEnzyme InhibitorsFuransImidazolesIndenesInterleukin-1betaKidneyLosartanMaleParaventricular Hypothalamic NucleusQuinoxalines4(2'-aminoethyl)amino-1,8-dimethylimidazo(1,2-a)quinoxalineAcetophenonesacetovanilloneAngiotensin II Type 1 Receptor BlockersCapsaicinEnzyme InhibitorsFuransImidazolesIndenesInterleukin-1betaLosartanN-(1,2,3,5,6,7-hexahydro-S-indacen-4-ylcarbamoyl)-4-(2-hydroxy-2-propanyl)-2-furansulfonamideQuinoxalinesReceptor, Angiotensin, Type 1Rela protein, ratSulfonamidesSuperoxidesTranscription Factor RelAAngiotensinBlood pressureInterleukinReactive oxygen speciesRenal reflexSympathetic activity

Identifiers

PMID32915316
OpenAlexW3084495409

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.