Evidence map›Paper›PMID 32859051›Full record

ArticleInternational journal of molecular sciences2020

Ameliorated Autoimmune Arthritis and Impaired B Cell Receptor-Mediated Ca

Esam Khanfar, Katalin Olasz, Fanni Gábris, Erzsébet Gajdócsi, Bálint Botz, Tamás Kiss, Réka Kugyelka, Tímea Berki, Péter Balogh, Ferenc Boldizsár

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in International journal of molecular sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.4field-weighted citation impact, top 40% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 10 citations in OpenAlex.

  1. Article
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  8. Research of Pathogenesis and Novel Therapeutics in Arthritis 2.0.International journal of molecular sciences · 2020
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 1 institution in 1 country.

Esam KhanfarDepartment of Immunology and Biotechnology, Medical School, University of Pécs, 7624 Pécs, Hungary.ORCID 0000-0001-5260-7937
Katalin OlaszDepartment of Immunology and Biotechnology, Medical School, University of Pécs, 7624 Pécs, Hungary.
Fanni GábrisDepartment of Immunology and Biotechnology, Medical School, University of Pécs, 7624 Pécs, Hungary.
Erzsébet GajdócsiDepartment of Immunology and Biotechnology, Medical School, University of Pécs, 7624 Pécs, Hungary.
Bálint BotzDepartment of Medical Imaging, Medical School, University of Pécs, 7624 Pécs, Hungary.
Tamás KissMolecular Pharmacology Research Group, János Szentágothai Research Centre and Centre for Neuroscience, University of Pécs, 7624 Pécs, Hungary.
Réka KugyelkaDepartment of Immunology and Biotechnology, Medical School, University of Pécs, 7624 Pécs, Hungary.ORCID 0000-0003-1784-1773
Tímea BerkiDepartment of Immunology and Biotechnology, Medical School, University of Pécs, 7624 Pécs, Hungary.
Péter BaloghDepartment of Immunology and Biotechnology, Medical School, University of Pécs, 7624 Pécs, Hungary.
Ferenc BoldizsárDepartment of Immunology and Biotechnology, Medical School, University of Pécs, 7624 Pécs, Hungary.ORCID 0000-0001-7831-1018
University of Pecs · HU

Funding

European Union, co-financed by the European Social Fund: Projects GINOP-2.3.2.-15-2016-00048European Union, co-financed by the European Social Fund: Projects "Comprehensive Development for Implementing Smart Specialization Strategies at the University of Pécs" EFOP-3.6.1.-16-2016-00004European Union, co-financed by the European Social Fund: Projects "PEPSYS - Complexity of peptide-signalization and its role in systemic diseases" GINOP 2.3.2-15-2016-00050European Union, co-financed by the European Social Fund: Projects "Stay Alive" EFOP-3.6.1-16-2016-00004János Bolyai Research Scholarship of The Hungarian Academy of Sciences BO/00501/19/5New National Excellence Program of the Ministry for Innovation and Technology ÚNKP-19-4-P-PTE-458
6 · The paper itself

Abstract

B cells play a crucial role in the pathogenesis of rheumatoid arthritis. In Nkx2-3-deficient mice (Nkx2-3

Indexed as

AggrecansAnimalsArthritis, ExperimentalArthritis, RheumatoidB-LymphocytesCalcium SignalingCells, CulturedCytokinesFemaleHomeodomain ProteinsHumansMiceMice, Inbred BALB CMice, KnockoutProtein DomainsSeverity of Illness IndexACAN protein, humanAggrecansCytokinesHomeodomain ProteinsNkx2.3 protein, mouseTranscription Factorsautoimmune arthritisB cell activationNkx2-3

Identifiers

PMID32859051
PMCPMC7503974
OpenAlexW3080304960

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.