Evidence map›Paper›PMID 32759269›Full record

ArticlePlant physiology2020

AtNBR1 Is a Selective Autophagic Receptor for AtExo70E2 in Arabidopsis.

Changyang Ji, Jun Zhou, Rongfang Guo, Youshun Lin, Chun-Hong Kung, Shuai Hu, Wing Yin Ng, Xiaohong Zhuang, Liwen Jiang

Open access · bronzeAbstract read
In one paragraph

Article in Plant physiology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 30 papers.

0numbers the graph read from it
0cells of the map it votes in
30citing papers in PubMed
3.8field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

30 citing papers in PubMed, 38 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Structural and Functional Characterization of EXPO-Derived Extracellular Vesicles in Plants.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026
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  15. Article
  16. Article
  17. Article
  18. NBR1: The archetypal selective autophagy receptor.The Journal of cell biology · 2022
    Review
  19. Review
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 2 countries.

Changyang JiCentre for Cell and Developmental Biology and State Key Laboratory of Agrobiotechnology, School of Life Sciences, the Chinese University of Hong Kong, Hong Kong, China ljiang@cuhk.edu.hk.ORCID 0000-0003-4399-8272
Jun ZhouCentre for Cell and Developmental Biology and State Key Laboratory of Agrobiotechnology, School of Life Sciences, the Chinese University of Hong Kong, Hong Kong, China.
Rongfang GuoCentre for Cell and Developmental Biology and State Key Laboratory of Agrobiotechnology, School of Life Sciences, the Chinese University of Hong Kong, Hong Kong, China.ORCID 0000-0003-0408-8700
Youshun LinCentre for Cell and Developmental Biology and State Key Laboratory of Agrobiotechnology, School of Life Sciences, the Chinese University of Hong Kong, Hong Kong, China.ORCID 0000-0003-2260-0202
Chun-Hong KungCentre for Cell and Developmental Biology and State Key Laboratory of Agrobiotechnology, School of Life Sciences, the Chinese University of Hong Kong, Hong Kong, China.ORCID 0000-0003-1244-5990
Shuai HuCentre for Cell and Developmental Biology and State Key Laboratory of Agrobiotechnology, School of Life Sciences, the Chinese University of Hong Kong, Hong Kong, China.ORCID 0000-0002-9737-2091
Wing Yin NgCentre for Cell and Developmental Biology and State Key Laboratory of Agrobiotechnology, School of Life Sciences, the Chinese University of Hong Kong, Hong Kong, China ljiang@cuhk.edu.hk.
Xiaohong ZhuangCentre for Cell and Developmental Biology and State Key Laboratory of Agrobiotechnology, School of Life Sciences, the Chinese University of Hong Kong, Hong Kong, China.ORCID 0000-0002-2480-8210
Liwen JiangCentre for Cell and Developmental Biology and State Key Laboratory of Agrobiotechnology, School of Life Sciences, the Chinese University of Hong Kong, Hong Kong, China ljiang@cuhk.edu.hk.ORCID 0000-0002-7829-1472
Chinese University of Hong Kong · HKMinistry of Education of the People's Republic of China · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Selective autophagy is a subcellular process whereby cytoplasmic materials are selectively sequestered into autophagosomes for subsequent delivery to the vacuole for degradation and recycling. Arabidopsis (

Indexed as

AutophagyArabidopsisArabidopsis ProteinsCarrier ProteinsVesicular Transport ProteinsArabidopsis ProteinsCarrier ProteinsExo70E2 protein, ArabidopsisNBR1 protein, ArabidopsisVesicular Transport Proteins

Identifiers

PMID32759269
PMCPMC7536653
OpenAlexW3048352105

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.