Evidence map›Paper›PMID 32708345›Full record

ReviewNutrients2020

Nutrients, Mitochondrial Function, and Perinatal Health.

Ameyalli M Rodríguez-Cano, Claudia C Calzada-Mendoza, Guadalupe Estrada-Gutierrez, Jonatan A Mendoza-Ortega, Otilia Perichart-Perera

Open access · goldAbstract readReview
In one paragraph

Review in Nutrients, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 34 papers.

0numbers the graph read from it
0cells of the map it votes in
34citing papers in PubMed
8.2field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

34 citing papers in PubMed, 54 citations in OpenAlex.

  1. Article
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  7. Review
  8. Transient fetal hyperglycemia reprograms adipogenesis, lipogenesis, and insulin signaling in zebrafish.Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie · 2026
    Article
  9. Observational
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  11. Article
  12. Review
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  15. Article
  16. Review
  17. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Ameyalli M Rodríguez-CanoSection for Postgraduate Studies and Research, Higher School of Medicine, Instituto Politécnico Nacional, Mexico City 11340, Mexico.ORCID 0000-0003-4477-0317
Claudia C Calzada-MendozaSection for Postgraduate Studies and Research, Higher School of Medicine, Instituto Politécnico Nacional, Mexico City 11340, Mexico.
Guadalupe Estrada-GutierrezInstituto Nacional de Perinatología Isidro Espinosa de los Reyes, Research Division; Montes Urales 800, Lomas de Virreyes, Mexico City 11000, Mexico.ORCID 0000-0001-9551-9021
Jonatan A Mendoza-OrtegaImmunobiochemistry Department, Instituto Nacional de Perinatología Isidro Espinosa de los Reyes, Montes Urales 800, Lomas de Virreyes, Mexico City 11000, Mexico.ORCID 0000-0003-0902-4351
Otilia Perichart-PereraNutrition and Bioprogramming Department, Instituto Nacional de Perinatología Isidro Espinosa de los Reyes, Montes Urales 800, Lomas de Virreyes, Mexico City 11000, Mexico.ORCID 0000-0003-1492-5293
Instituto Nacional de Perinatología · MXInstituto Politécnico Nacional · MX

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Mitochondria are active independent organelles that not only meet the cellular energy requirement but also regulate central cellular activities. Mitochondria can play a critical role in physiological adaptations during pregnancy. Differences in mitochondrial function have been found between healthy and complicated pregnancies. Pregnancy signifies increased nutritional requirements to support fetal growth and the metabolism of maternal and fetal tissues. Nutrient availability regulates mitochondrial metabolism, where excessive macronutrient supply could lead to oxidative stress and contribute to mitochondrial dysfunction, while micronutrients are essential elements for optimal mitochondrial processes, as cofactors in energy metabolism and/or as antioxidants. Inadequate macronutrient and micronutrient consumption can result in adverse pregnancy outcomes, possibly through mitochondrial dysfunction, by impairing energy supply, one-carbon metabolism, biosynthetic pathways, and the availability of metabolic co-factors which modulate the epigenetic processes capable of establishing significant short- and long-term effects on infant health. Here, we review the importance of macronutrients and micronutrients on mitochondrial function and its influence on maternal and infant health.

Indexed as

Infant HealthMaternal HealthMicronutrientsNutrientsNutritional RequirementsEatingEnergy MetabolismEpigenesis, GeneticFemaleHumansInfant, NewbornInfant Nutritional Physiological PhenomenaMaleMaternal-Fetal ExchangeMaternal Nutritional Physiological PhenomenaMitochondriaMicronutrientsNutrientsVitaminsdietmacronutrientsmicronutrientsmineralspregnancyvitamins

Identifiers

PMID32708345
PMCPMC7401276
OpenAlexW3044982937

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.