Evidence map›Paper›PMID 32670503›Full record

ArticleComputational and structural biotechnology journal2020

Exploring the structural basis of conformational heterogeneity and autoinhibition of human cGMP-specific protein kinase Iα through computational modelling and molecular dynamics simulations.

Arooma Maryam, Rana Rehan Khalid, Sundeep Chaitanya Vedithi, Abdulilah Ece, Suleyman Selim Çınaroğlu, Abdul Rauf Siddiqi, Tom L Blundell

Open access · goldAbstract read
In one paragraph

Article in Computational and structural biotechnology journal, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.8field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 10 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 4 institutions in 3 countries.

Arooma MaryamDepartment of Biosciences, COMSATS University Islamabad (CUI), Park Road, Islamabad 4550, Pakistan.
Rana Rehan KhalidDepartment of Biosciences, COMSATS University Islamabad (CUI), Park Road, Islamabad 4550, Pakistan.
Sundeep Chaitanya VedithiDepartment of Biochemistry, University of Cambridge, 80 Tennis Court Rd., Cambridge CB2 1GA, UK.
Abdulilah EceDepartment of Pharmaceutical Chemistry, Faculty of Pharmacy, Biruni University, Istanbul 34010, Turkey.
Suleyman Selim ÇınaroğluDepartment of Biochemistry, University of Oxford, South Parks Road, Oxford OX1 3QU, UK.
Abdul Rauf SiddiqiDepartment of Biosciences, COMSATS University Islamabad (CUI), Park Road, Islamabad 4550, Pakistan.
Tom L BlundellDepartment of Biochemistry, University of Cambridge, 80 Tennis Court Rd., Cambridge CB2 1GA, UK.
COMSATS University Islamabad · PKUniversity of Cambridge · GBBiruni University · TRUniversity of Oxford · GB

Funding

Wellcome Trust 200814/Z/16/Z
6 · The paper itself

Abstract

Protein kinase Iα (PKGIα) is a pivotal cyclic guanosine monophosphate (cGMP) signalling protein. Major steps related to the structural plasticity of PKGIα have been inferred but the structural aspects of the auto-inhibition and multidomain tertiary organization of human PKGIα in active and inactive form are not clear. Here we combine computational comparative modelling, protein-protein docking and molecular dynamics (MD) simulations to investigate structural details of the repressed state of the catalytic domain of PKGIα. Exploration of the potential inhibitory conformation of the auto-inhibitory domain (AI) within the catalytic cleft reveals that the pseudo-substrate motif binds with residues of the glycine rich loop and substrate-binding lobe. Dynamic changes as a result of coupling of the catalytic and AI domains are also investigated. The three-dimensional homodimeric models of PKGIα in the active and inactive state indicate that PKGIα in its inactive-state attains a compact globular structure where cyclic nucleotide binding (CNB-A/B) domains are buried, whereas the catalytic domains are inaccessible with their substrate-binding pockets facing the N-terminal of CNB-A. Contrary to this, the active-state model of PKGIα shows an extended conformation where CNB-A/B domains are slightly rearranged and the catalytic domains of homodimer flanking the C-terminal with their substrate binding lobes free to entrap downstream proteins. These findings are consistent with previously reported static images of the multidomain organization of PKGIα. Structural insights pertaining to the conformational heterogeneity and auto-inhibition of PKGIα provided in this study may help to understand the dynamics-driven effective regulation of PKGIα.

Indexed as

Auto-inhibitory domain (AI)Cyclic guanosine monophosphate (cGMP)Cyclic Nucleotide binding domain-A (CNB-A)Protein kinase Iα (PKGIα)

Identifiers

PMID32670503
PMCPMC7334484
OpenAlexW3034548792

What OpenQuestion holds

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LicenceCC BY-NC-ND
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.