ReviewEye and brain2020
Neuro-ophthalmic Manifestations of Wernicke Encephalopathy.
Review in Eye and brain, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
28 citing papers in PubMed, 42 citations in OpenAlex.
- Article
- Article
- Ophthalmoplegia and gaze-evoked nystagmus in Wernicke encephalopathy.CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne · 2026Article
- Astrocytopathy in Wernicke Encephalopathy and Neuromyelitis Optica Spectrum Disorder. Pathogenic Differences With Occasional Clinical and Neuroimaging Overlap. A Review.Current neurology and neuroscience reports · 2026Review
- Not All Who Stagger Are Drunk: Ocular and Gait Abnormalities in a Young Adult.Journal of the American College of Emergency Physicians open · 2026Article
- Associations Between Nutritional Factors, Obesity and Ocular Diseases: A Narrative Literature Review.Nutrients · 2025Review
- Thiamine Deficiency and Brain Injury: Neuroanatomical Changes in the Wernicke-Korsakoff Syndrome.Cureus · 2025Review
- Article
- Swept-Source Optical Coherence Tomography in the Diagnosis and Monitoring of Optic Nerve Neuropathy in Patients with Wernicke's Encephalopathy Due to Hyperemesis Gravidarum.Journal of clinical medicine · 2025Review
- Concurrent Wernicke Encephalopathy and Posterior Reversible Encephalopathy Syndrome Following Gastric Sleeve Surgery.Ochsner journal · 2025Article
- Wernicke encephalopathy: a mini review of the clinical spectrum, atypical manifestations, and diagnostic challenges.Frontiers in neurology · 2025Review
- Reversible Bilateral Vision Loss: An Unusual Presentation of Wernicke-Korsakoff Syndrome.Cureus · 2024Article
- Article
- Wernicke encephalopathy with atypical imaging findings in a depressed patient: A case report.Radiology case reports · 2024Article
- Ocular manifestations of liver disease: an important diagnostic aid.International ophthalmology · 2024Review
- Nystagmus and Abducens Nerve Palsy as an Early Presentation of Non-alcoholic Wernicke Encephalopathy.Cureus · 2024Article
- An overview of neuro-ophthalmic disorders at Jenna Ophthalmic Center, Baghdad, Iraq (2021-2022).Journal of medicine and life · 2024Observational
- Wernicke's Encephalopathy and Serotonin Syndrome: A Case Report of Overlapping Pathologies.The Journal of pharmacy technology : jPT : official publication of the Association of Pharmacy Technicians · 2023Article
- Uncommon bilateral optic neuropathy in Wernicke's encephalopathy complicating gravidarum hyperemesis.La Tunisie medicale · 2023Article
- Association Between Vitamin Deficiencies and Ophthalmological Conditions.Clinical ophthalmology (Auckland, N.Z.) · 2023Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors at 2 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Wernicke encephalopathy (WE) is a life-threatening but reversible syndrome resulting from acute thiamine deficiency that is frequently overlooked and underdiagnosed. It is classically characterized by a triad of ocular dysfunction, ataxia, and altered mental status. However, less than 1/3 patients have the complete triad, so it is crucial to have a high index of suspicion. Awareness of the early signs of WE is essential to prevent clinical progression, as patients with the full triad already have a profoundly thiamine-deficient state. This review highlights the neuro-ophthalmic manifestations of WE to guide the clinician in identifying the condition. In addition, we provide an update regarding the clinical characteristics, pathophysiology, neuroimaging and laboratory findings, treatment options, and prognosis of WE.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.