Evidence map›Paper›PMID 32613603›Full record

ArticleNeurotoxicity research2020

Genistein and Galantamine Combinations Decrease β-Amyloid Peptide

Willian Orlando Castillo, Nilza Velasco Palomino, Catarina Satie Takahashi, Silvana Giuliatti

Abstract read
PubMed Publisher
In one paragraph

Article in Neurotoxicity research, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.9field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 21 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Willian Orlando CastilloDepartment of Biology, Faculty of Natural Sciences and Education, University of Cauca, Cra 2 No 2N-57, Popayán, 19003, Colombia. wocastillo@unicauca.edu.co.ORCID https://orcid.org/0000-0001-9138-1248
Nilza Velasco PalominoDepartment of Biology, Faculty of Natural Sciences and Education, University of Cauca, Cra 2 No 2N-57, Popayán, 19003, Colombia.ORCID https://orcid.org/0000-0003-1494-9674
Catarina Satie TakahashiDepartment of Genetics, Ribeirão Preto Medical School, University of São Paulo-USP, Ribeirão Preto, 14090-900, Brazil.ORCID https://orcid.org/0000-0003-3367-539X
Silvana GiuliattiDepartment of Genetics, Ribeirão Preto Medical School, University of São Paulo-USP, Ribeirão Preto, 14090-900, Brazil.ORCID https://orcid.org/0000-0002-7520-6420
Universidade de Ribeirão Preto · BRUniversity of Cauca · CO

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Alzheimer's disease (AD) is the primary dementia-causing disease worldwide, involving a multifactorial combination of environmental, genetic, and epigenetic factors, with essential participation of age and sex. Biochemically, AD is characterized by the presence of abnormal deposition of beta amyloid peptide (Aβ

Indexed as

Amyloid beta-PeptidesApoptosisCholinesterase InhibitorsGalantamineGenisteinHumansMolecular Docking SimulationNeuroprotective AgentsOxidative StressAmyloid beta-PeptidesCholinesterase InhibitorsGalantamineGenisteinNeuroprotective AgentsAlzheimer’s diseaseEstrogenMolecular dockingNeurotoxicityOxidative stressSynergistic

Identifiers

PMID32613603
OpenAlexW3040165205

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.