Evidence map›Paper›PMID 32613277›Full record

ArticlePediatric nephrology (Berlin, Germany)2020

Evaluation of phenotypic and genotypic features of children with distal kidney tubular acidosis.

Bahriye Atmis, Derya Cevizli, Engin Melek, Atil Bisgin, Ilker Unal, Ali Anarat, Aysun K Bayazit

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Article in Pediatric nephrology (Berlin, Germany), 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.0field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 16 citations in OpenAlex.

  1. Article
  2. Etiology and outcomes of primary renal tubular acidosis.Pediatric nephrology (Berlin, Germany) · 2025
    Article
  3. Article
  4. Primary Distal Renal Tubular Acidosis: Toward an Optimal Correction of Metabolic Acidosis.Clinical journal of the American Society of Nephrology : CJASN · 2024
    Review
  5. Review
  6. Review
  7. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Bahriye AtmisDepartment of Pediatric Nephrology, Cukurova University Faculty of Medicine, Adana, Turkey. bahriyeatmis@gmail.com.
Derya CevizliDepartment of Pediatric Nephrology, Cukurova University Faculty of Medicine, Adana, Turkey.
Engin MelekDepartment of Pediatric Nephrology, Cukurova University Faculty of Medicine, Adana, Turkey.
Atil BisginDepartment of Medical Genetics, Faculty of Medicine, Cukurova University, Adana, Turkey.
Ilker UnalDepartment of Biostatistics, Cukurova University Faculty of Medicine, Adana, Turkey.
Ali AnaratDepartment of Pediatric Nephrology, Cukurova University Faculty of Medicine, Adana, Turkey.
Aysun K BayazitDepartment of Pediatric Nephrology, Cukurova University Faculty of Medicine, Adana, Turkey.
Cukurova University · TR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundThe present study aimed to assess genotype-phenotype correlations with long-term prognosis in children with distal kidney tubular acidosis (dKTA). The kidney function of children with dKTA could be impaired in the long-term.

methodsThirty-one children with dKTA from 23 families were included in the present study. Demographic features, growth parameters, clinical manifestations, follow-up results, and genetic analysis results of the patients were recorded.

resultsEighteen children (58.1%) were male. The median age at diagnosis was 3 months. The median follow-up period was 77 months and the longest was 23.5 years. Eight (28.8%) patients had chronic kidney disease (CKD) stage 2 or 3. Three patients aged 24, 23, and 19 years had CKD stage 3 with an estimated glomerular filtration rate of 54, 57, and 48 mL/min/1.73 m

conclusionPatients with dKTA usually have a good clinical prognosis in childhood with appropriate treatment; however, dRTA is characterized by deterioration of kidney function in adulthood, particularly after puberty.

Indexed as

Acidosis, Renal TubularAnion Exchange Protein 1, ErythrocyteChild, PreschoolDisease ProgressionFemaleGenetic MarkersGlomerular Filtration RateHumansInfantMaleMutationRenal Insufficiency, ChronicVacuolar Proton-Translocating ATPasesAnion Exchange Protein 1, ErythrocyteATP6V0A4 protein, humanATP6V1B1 protein, humanGenetic MarkersSLC4A1 protein, humanVacuolar Proton-Translocating ATPasesChildrenDistal kidney tubular acidosisGenetic analysisLong-term prognosis

Identifiers

PMID32613277
OpenAlexW3038225117

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.