Evidence map›Paper›PMID 32550259›Full record

ReviewInternational journal of cardiology. Heart & vasculature2020

Coronavirus disease 2019 and cardiovascular system: A narrative review.

Felix Kwenandar, Karunia Valeriani Japar, Vika Damay, Timotius Ivan Hariyanto, Michael Tanaka, Nata Pratama Hardjo Lugito, Andree Kurniawan

Open access · goldAbstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in International journal of cardiology. Heart & vasculature, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 74 papers, 14 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
74citing papers in PubMed, 14 pooled it
3.0field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

74 citing papers in PubMed, 14 syntheses or guidelines pooled it, 126 citations in OpenAlex.

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  8. Statin and outcomes of coronavirus disease 2019 (COVID-19): A systematic review, meta-analysis, and meta-regression.Nutrition, metabolism, and cardiovascular diseases : NMCD · 2021 · on this map
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  15. Article
  16. Evaluation of myocardial work and exercise capacity in patients recovered from the severe form of COVID-19.International journal of cardiology. Cardiovascular risk and prevention · 2024
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14 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Felix KwenandarFaculty of Medicine, Pelita Harapan University, Banten, Indonesia.
Karunia Valeriani JaparFaculty of Medicine, Pelita Harapan University, Banten, Indonesia.
Vika DamayFaculty of Medicine, Pelita Harapan University, Banten, Indonesia.
Timotius Ivan HariyantoFaculty of Medicine, Pelita Harapan University, Banten, Indonesia.
Michael TanakaFaculty of Medicine, Pelita Harapan University, Department of Cardiology, Siloam Hospital Lippo Village, Karawaci, Banten, Indonesia.
Nata Pratama Hardjo LugitoFaculty of Medicine, Pelita Harapan University, Department of Internal Medicine, Siloam Hospital Lippo Village, Karawaci, Banten, Indonesia.
Andree KurniawanFaculty of Medicine, Pelita Harapan University, Department of Internal Medicine, Siloam Hospital Lippo Village, Karawaci, Banten, Indonesia.
Pelita Harapan University · ID

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

At the end of 2019, a viral pneumonia disease called coronavirus disease 2019 (COVID-19), which is caused by severe acute respiratory syndrome coronavirus-2 (SARS-CoV2), emerged in Wuhan, China. This novel disease rapidly spread at an alarming rate that as a result, it has now been declared pandemic by the World Health Organization. Although this infective disease is mostly characterized by respiratory tract symptoms, increasing numbers of evidence had shown considerable amounts of patients with cardiovascular involvements and these were associated with higher mortality among COVID-19 patients. Cardiac involvement as a possible late phenomenon of the viral respiratory infection is an issue that should be anticipated in patients with COVID-19. Cardiovascular manifestation in COVID-19 patients include myocardial injury (MI), arrhythmias, cardiac arrests, heart failure and coagulation abnormality, ranging from 7.2% up to 33%. The mechanism of cardiac involvement in COVID-19 patients involves direct injury to myocardial cells mediated by angiotensin-converting enzyme 2 (ACE2) receptors as suggested by some studies, while the other studies suggest that systemic inflammation causing indirect myocyte injury may also play a role. Combination of proper triage, close monitoring, and avoidance of some drugs that have cardiovascular toxicity are important in the management of cardiovascular system involvement in COVID-19 patients. The involvement of the cardiovascular system in COVID-19 patients is prevalent, variable, and debilitating. Therefore, it requires our attention and comprehensive management.

Indexed as

ACE2ACE2, Angiotensin Converting Enzyme-2ARDS, Acute respiratory distress syndromeCardiovascularCFR, Case-fatality rateCK-MB, Creatine Kinase Myocardial BandCoronavirusCOVID-19COVID-19, Coronavirus Disease 2019CPVT, Catecholaminergic polymorphic ventricular tachycardiaCVD, Cardiovascular diseaseDIC, Disseminated intravascular coagulationFDP, Fibrin degradation productsHs-cTnI, High-sensitive cardiac troponin ILV, Left VentricleMI, Myocardial InfarctionNT-proBNP, N-terminal prohormone of brain natriuretic peptidePCI, Percutaneous coronary interventionSARS-CoV-2SARS-CoV-2, Severe Acute Respiratory Syndrome Coronavirus 2TnT, Troponin TVTE, Venous thromboembolism

Identifiers

PMID32550259
PMCPMC7266760
OpenAlexW3033599099

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.