Evidence map›Paper›PMID 32489565›Full record

ArticleIranian journal of basic medical sciences2020

High proviral load of human T cell lymphotropic virus type-1 facilitates coronary artery diseases.

Farnaz Mozayeni, Seyed Abdolrahim Rezaee, Farahzad Jabbari Azad, Mahmoud Shabestari, Reza Faridhosseini, Houshang Rafatpanah, Hadis Yousefzadeh, Yousef Ali Garivani, Lida Jarahi, Narges Valizadeh and 4 more

Open access · greenAbstract read
In one paragraph

Article in Iranian journal of basic medical sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact, top 99% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 2 institutions in 1 country.

Farnaz MozayeniAllergy Research Center, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Seyed Abdolrahim RezaeeImmunology Research Center, Inflammation and Inflammatory Disease Division, Mashhad University of Medical Sciences, Mashhad, Iran.
Farahzad Jabbari AzadAllergy Research Center, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Mahmoud ShabestariDepartment of Cardiology, Emam Reza Hospital, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Reza FaridhosseiniAllergy Research Center, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Houshang RafatpanahImmunology Research Center, Inflammation and Inflammatory Disease Division, Mashhad University of Medical Sciences, Mashhad, Iran.
Hadis YousefzadehImmunology Research Center, Bu Ali Research Institute, Student Research Committee, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Yousef Ali GarivaniDepartment of Cardiology, Emam Reza Hospital, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Lida JarahiAddiction Research Center, Mashhad University of Medical Sciences, Faculty of Medicine, Mashhad, Iran.
Narges ValizadehImmunology Research Center, Inflammation and Inflammatory Disease Division, Mashhad University of Medical Sciences, Mashhad, Iran.
Faezah SabetImmunology Research Center, Inflammation and Inflammatory Disease Division, Mashhad University of Medical Sciences, Mashhad, Iran.
Sharare MoshirahmadiDepartment of Cardiology, Emam Reza Hospital, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Fatemeh Sadat MohammadiAllergy Research Center, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Mohammad ShabestariDepartment of Cardiology, Emam Reza Hospital, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Mashhad University of Medical Sciences · IREmam Reza Hospital · IR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectivesCoronary artery disease (CAD) is known as a life threatening disease, worldwide. In this study the role of HTLV-1 infection was evaluated on cardiac involvement in an endemic region of northeastern Iran. MATERIALS AND

methodsSerologic and molecular tests for HTLV-1 infection were carried out in subjects who had coronary angiography. A real-time PCR, TaqMan method, to quantify HTLV-1 proviral load (PVL), and routine hematological and biochemical tests were performed for study subjects.

resultsTwenty nine patients were HTLV-1+CAD+ and 13 cases were HTLV-1+CAD-. Although, there were no significant differences for risk factors like FBS, HDL, triglyceride, systolic and diastolic blood pressure (Cbp, Dbp), waist circumference (WC), hip circumference (WL), cholesterol (

conclusionTaken together, in endemic area, HTLV-1 infection, more likely is a facilitating factor for heart complications and the high HTLV-1 PVL might affect CAD manifestations.

Indexed as

Cardiac involvementCoronary artery diseaseHTLV-1HTLV-1 Proviral loadReal-time PCR

Identifiers

PMID32489565
PMCPMC7239420
OpenAlexW3004744345

What OpenQuestion holds

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LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.