Evidence map›Paper›PMID 32482922›Full record

ArticleJournal of genetics2020

CG/CA genotypes represent novel markers in the

Leila Esmaeli Chamgordani, Nasim Ebrahimi, Farzane Amirmahani, Sadeq Vallian

Abstract read
PubMed
In one paragraph

Article in Journal of genetics, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.7field-weighted citation impact, top 31% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 5 citations in OpenAlex.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Leila Esmaeli ChamgordaniGenetics Division, Faculty of Biological Sciences and Technologies, Department of Cellular and Molecular Biology and Microbiology, University of Isfahan, Isfahan, Iran. svallian@sci.ui.ac.ir.
Nasim Ebrahimi
Farzane Amirmahani
Sadeq Vallian
University of Isfahan · IR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Nephrotic syndrome (NS) is considered as a primary disease of the kidney that represents a heterogeneous group of glomerular disorders occurring mainly in children. It is generally divided into steroid-sensitive and steroid-resistant forms, depending upon the patient's response to steroid therapy. Among the genes involved, the

Indexed as

AllelesBiomarkersChildFemaleGene FrequencyGenotypeHaplotypesHeterozygoteHumansIntracellular Signaling Peptides and ProteinsIranMaleMembrane ProteinsMutationNephrotic SyndromePolymerase Chain ReactionBiomarkersIntracellular Signaling Peptides and ProteinsMembrane ProteinsNPHS2 protein

Identifiers

PMID32482922
OpenAlexW3021460858

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.