Evidence map›Paper›PMID 32477937›Full record

ArticleFrontiers in oncology2020

Protective Role of Decorin in Primary Hepatocellular Carcinoma.

Andrea Reszegi, Zsolt Horváth, Hajnalka Fehér, Barnabás Wichmann, Péter Tátrai, Ilona Kovalszky, Kornélia Baghy

Open access · goldAbstract read
In one paragraph

Article in Frontiers in oncology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 26 papers.

0numbers the graph read from it
0cells of the map it votes in
26citing papers in PubMed
2.6field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

26 citing papers in PubMed, 36 citations in OpenAlex.

  1. Article
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  7. Hypoxia-Induced Suppression of FAM99A and FAM99B Contributes to the Development and Glucose Metabolic Reprogramming of Hepatocellular Carcinoma.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2025
    Article
  8. Decorin: matrix-based pan-cancer tumor suppressor.Molecular and cellular biochemistry · 2025
    Review
  9. Article
  10. Review
  11. Article
  12. Review
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  14. Article
  15. Article
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  17. Review
  18. Isotretinoin and Thalidomide Down-RegulateMolecules (Basel, Switzerland) · 2021
    Article
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Andrea Reszegi1st Department of Pathology and Experimental Cancer Research, Semmelweis University, Budapest, Hungary.
Zsolt Horváth1st Department of Pathology and Experimental Cancer Research, Semmelweis University, Budapest, Hungary.
Hajnalka Fehér1st Department of Pathology and Experimental Cancer Research, Semmelweis University, Budapest, Hungary.
Barnabás Wichmann2nd Department of Internal Medicine, Semmelweis University, Budapest, Hungary.
Péter TátraiSolvo Biotechnology, Budapest, Hungary.
Ilona Kovalszky1st Department of Pathology and Experimental Cancer Research, Semmelweis University, Budapest, Hungary.
Kornélia Baghy1st Department of Pathology and Experimental Cancer Research, Semmelweis University, Budapest, Hungary.
Semmelweis University · HUSolvo Biotechnology (Hungary) · HU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hepatocellular carcinoma (HCC) represents one of the most frequent type of primary liver cancers. Decorin, a small leucine-rich proteoglycan of the extracellular matrix, represents a powerful tumor cell growth and migration inhibitor by hindering receptor tyrosine kinases and inducing p21

Indexed as

decorin (DCN)extracellular matrix (ECM)hepatocarcinogenesishepatocellular carcinomaproteoglycan (PG)

Identifiers

PMID32477937
PMCPMC7235294
OpenAlexW3022661528

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.