Evidence map›Paper›PMID 32472332›Full record

ReviewPflugers Archiv : European journal of physiology2020

TASK channels: channelopathies, trafficking, and receptor-mediated inhibition.

Masumi Inoue, Hidetada Matsuoka, Keita Harada, Go Mugishima, Masaki Kameyama

Abstract readReview
PubMed Publisher
In one paragraph

Review in Pflugers Archiv : European journal of physiology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.0field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 21 citations in OpenAlex.

  1. Dynamic regulation of TASK-1 channels under cellular stress.The journal of physiological sciences : JPS · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Masumi InoueDepartment of Cell and Systems Physiology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, 807-8555, Japan. minoue@med.uoeh-u.ac.jp.ORCID 0000-0003-4180-5238
Hidetada MatsuokaDepartment of Cell and Systems Physiology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, 807-8555, Japan.
Keita HaradaDepartment of Cell and Systems Physiology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, 807-8555, Japan.
Go MugishimaDepartment of Cell and Systems Physiology, University of Occupational and Environmental Health School of Medicine, Kitakyushu, 807-8555, Japan.
Masaki KameyamaDepartment of Physiology, Kagoshima University Graduate School of Medical and Dental Sciences, Kagoshima, 890-8544, Japan.
University of Occupational and Environmental Health Japan · JPKagoshima University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

TWIK-related acid-sensitive K

Indexed as

AnimalsChannelopathiesHumansNerve Tissue ProteinsPhosphorylationPotassium Channels, Tandem Pore DomainProtein TransportReceptors, G-Protein-CoupledSignal TransductionNerve Tissue ProteinsPotassium Channels, Tandem Pore Domainpotassium channel subfamily K member 3Receptors, G-Protein-CoupledAdrenal medullary chromaffin cellp11PC12 cellTASK1TASK3

Identifiers

PMID32472332
OpenAlexW3029135441

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.