Evidence map›Paper›PMID 32457093›Full record

ArticlePlant physiology2020

Loss of the Acetyltransferase NAA50 Induces Endoplasmic Reticulum Stress and Immune Responses and Suppresses Growth.

Matthew Neubauer, Roger W Innes

Open access · bronzeAbstract read
In one paragraph

Article in Plant physiology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
1.3field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 23 citations in OpenAlex.

  1. Article
  2. Article
  3. High Overexpression ofInternational journal of molecular sciences · 2024
    Article
  4. Review
  5. Natural Allelic Variations ofInternational journal of molecular sciences · 2024
    Article
  6. Article
  7. Review
  8. Article
  9. HYPK promotes the activity of theScience advances · 2022
    Article
  10. Article
  11. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Matthew NeubauerDepartment of Biology, Indiana University, Bloomington, Indiana 47405.ORCID 0000-0001-6579-2326
Roger W InnesDepartment of Biology, Indiana University, Bloomington, Indiana 47405 rinnes@indiana.edu.ORCID 0000-0001-9634-1413
Indiana University Bloomington · US

Funding

Programmed Cell Death and Disease Resistance in ArabidopsisR01GM063761 · NIGMS · TRUSTEES OF INDIANA UNIVERSITY · PI INNES, ROGER W. · 2002 to 2013
$3.5M
NIGMS NIH HHS R01 GM063761
6 · The paper itself

Abstract

Stress signaling in plants is carefully regulated to ensure proper development and reproductive fitness. Overactive defense signaling can result in dwarfism as well as developmental defects. In addition to requiring a substantial amount of energy, plant stress responses place a burden upon the cellular machinery, which can result in the accumulation of misfolded proteins and endoplasmic reticulum (ER) stress. Negative regulators of stress signaling, such as

Indexed as

AcetyltransferasesArabidopsisArabidopsis ProteinsEndoplasmic Reticulum StressGene Expression Regulation, PlantPlants, Genetically ModifiedAcetyltransferasesArabidopsis ProteinsEDR1 protein, Arabidopsis

Identifiers

PMID32457093
PMCPMC7401112
OpenAlexW3028665039

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.