Evidence map›Paper›PMID 32422904›Full record

ArticleAntioxidants (Basel, Switzerland)2020

Brain Transcriptome Analysis Links Deficiencies of Stress-Responsive Proteins to the Pathomechanism of Kii ALS/PDC.

Satoru Morimoto, Mitsuru Ishikawa, Hirotaka Watanabe, Miho Isoda, Masaki Takao, Shiho Nakamura, Fumiko Ozawa, Yoshifumi Hirokawa, Shigeki Kuzuhara, Hideyuki Okano and 1 more

Open access · goldAbstract read
In one paragraph

Article in Antioxidants (Basel, Switzerland), 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.6field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 9 citations in OpenAlex.

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  5. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 4 institutions in 1 country.

Satoru MorimotoDepartment of Physiology, School of Medicine, Keio University, Tokyo 160-8582, Japan.
Mitsuru IshikawaDepartment of Physiology, School of Medicine, Keio University, Tokyo 160-8582, Japan.
Hirotaka WatanabeDepartment of Physiology, School of Medicine, Keio University, Tokyo 160-8582, Japan.
Miho IsodaDepartment of Physiology, School of Medicine, Keio University, Tokyo 160-8582, Japan.
Masaki TakaoDepartment of Clinical Laboratory, National Center of Neurology and Psychiatry (NCNP), Tokyo 187-0031, Japan.
Shiho NakamuraDepartment of Physiology, School of Medicine, Keio University, Tokyo 160-8582, Japan.
Fumiko OzawaDepartment of Physiology, School of Medicine, Keio University, Tokyo 160-8582, Japan.
Yoshifumi HirokawaDepartment of Oncologic Pathology, School of Medicine, Mie University, Mie 514-8507, Japan.
Shigeki KuzuharaGraduate School of Health Science, Suzuka University of Medical Science, Mie 510-0293, Japan.
Hideyuki OkanoDepartment of Physiology, School of Medicine, Keio University, Tokyo 160-8582, Japan.
Yasumasa KokuboKii ALS/PDC Research Center, Graduate School of Regional Innovation Studies, Mie University, Mie 514-8507, Japan.
Keio University · JPMie University · JPNational Center of Neurology and Psychiatry · JPSuzuka University of Medical Science · JP

Funding

Japan Agency for Medical Research and Development 17ek0109139h0003Japan Agency for Medical Research and Development 19bm0804003h0003Japan Foundation for Neuroscience and Mental Health noneJapan Intractable Diseases (Nanbyo) Research Foundation noneJapan Society for the Promotion of Science 16H06277Ministry of Education, Culture, Sports, Science and Technology 15J03921Ministry of Education, Culture, Sports, Science and Technology 15K09634Ministry of Education, Culture, Sports, Science and Technology 17H01689Ministry of Education, Culture, Sports, Science and Technology 18K07368Ministry of Education, Culture, Sports, Science and Technology 18K07514Ministry of Education, Culture, Sports, Science and Technology 18KK0239Ministry of Education, Culture, Sports, Science and Technology 19K17002Ministry of Education, Culture, Sports, Science and Technology 25305030Ministry of Health, Labour and Welfare 21210301the Mie Medical Fund nonethe Research Committee of CNS Degenerative Diseases H29-Nanchi- Ippan-085
6 · The paper itself

Abstract

Amyotrophic lateral sclerosis and Parkinsonism-dementia complex (ALS/PDC) is a unique endemic neurodegenerative disease, with high-incidence foci in Kii Peninsula, Japan. To gather new insights into the pathological mechanisms underlying Kii ALS/PDC, we performed transcriptome analyses of patient brains. We prepared frozen brains from three individuals without neurodegenerative diseases, three patients with Alzheimer's disease, and 21 patients with Kii ALS/PDC, and then acquired microarray data from cerebral gray and white matter tissues. Microarray results revealed that expression levels of genes associated with heat shock proteins, DNA binding/damage, and senescence were significantly altered in patients with ALS/PDC compared with healthy individuals. The RNA expression pattern observed for ALS-type brains was similar to that of PDC-type brains. Additionally, pathway and network analyses indicated that the molecular mechanism underlying ALS/PDC may be associated with oxidative phosphorylation of mitochondria, ribosomes, and the synaptic vesicle cycle; in particular, upstream regulators of these mechanisms may be found in synapses and during synaptic trafficking. Furthermore, phenotypic differences between ALS-type and PDC-type were observed, based on HLA haplotypes. In conclusion, determining the relationship between stress-responsive proteins, synaptic dysfunction, and the pathogenesis of ALS/PDC in the Kii peninsula may provide new understanding of this mysterious disease.

Indexed as

Amyotrophic lateral sclerosis (ALS)heat shock proteinsHLA typingKii peninsulamitochondriaParkinsonism-dementia complex (PDC)stress-responsive proteinssynaptic traffickingtranscriptome analysis

Identifiers

PMID32422904
PMCPMC7278732
OpenAlexW3024397463

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.